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These are hypotheses against facts;

This is due to nanotechnology.

Why are you refusing to investigate this?

https://jnanobiotechnology.biomedcentral.com/articles/10.1186/1477-3155-11-26

Abstract

Interaction of nanoparticles with proteins is the basis of nanoparticle bio-reactivity. This interaction gives rise to the formation of a dynamic nanoparticle-protein corona. The protein corona may influence cellular uptake, inflammation, accumulation, degradation and clearance of the nanoparticles. Furthermore, the nanoparticle surface can induce conformational changes in adsorbed protein molecules which may affect the overall bio-reactivity of the nanoparticle.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5617637/

Etc.

https://outraged.substack.com/p/evidence-of-crimes-against-humanity

https://outraged.substack.com/p/chuck-norris-against-quantum-dots

https://outraged.substack.com/p/2-nobel-prizes-for-tagging-humans

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https://www.sciencedirect.com/science/article/abs/pii/S0009898121001376 Oxidative stress in vascular calcification

https://www.frontiersin.org/articles/10.3389/fphar.2022.858160/full

Reactive Oxygen Species in Cardiovascular Calcification: Role of Medicinal Plants

etc.

One of the main mechanisms of nanotechnology toxicity is oxidative stress.

In addition, several typical mechanisms underlying GFN toxicity have been revealed, for instance, physical destruction, oxidative stress, DNA damage, inflammatory response, apoptosis, autophagy, and necrosis. In these mechanisms, (toll-like receptors-) TLR-, transforming growth factor β- (TGF-β-) and tumor necrosis factor-alpha (TNF-α) dependent-pathways are involved in the signalling pathway network, and oxidative stress plays a crucial role in these pathways.

https://particleandfibretoxicology.biomedcentral.com/articles/10.1186/s12989-016-0168-y

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