Jan 24Liked by Walter M Chesnut

Walter, I'm an MD and routinely 'wage war' on a medical social media site called Doximity. I routinely read your substack along with many others of similar ilk. I do my best to learn from &, in a way, validate yours and others findings and I appreciate your and others herculean efforts to bring education and truth to light. My private war is one of extreme frustration and has become more of a cathartic exercise rather than a crusade to educate others. The level of obstinance, arrogance, political zeal and unfortunately, frank stupidity, that is exhibited on all that is COVID (by those who should know better) is utterly astonishing, infuriating and depressing. As you so eloquently show, the major points of understanding covid and the fraud that is the 'vaccine' are covered in the 'basic science' years of the first two years of med school. In short, they are forgotten and/or ignored in a very high percentage of my clinical brethren. Keep up the fantastic work and many thanks for your efforts.

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"When cells lose the tools they make to do their jobs, they lose their identity."

For those of us who struggle greatly w science it's priceless to have a clear grasp of the concept and idea we can repeat w confidence citing slam dunk source, many thanks Walter! <3

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I am so impressed by your perfecting your understanding and examining this process, and very grateful you are sharing it.

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"Some cells go senescent, some proliferate rapidly (cancer), some no longer function."

Here is the definition of senescence from cancer.gov:

Senesence (seh-Neh-sents) The process of growing old. In biology, senescence is a process by which a cell ages and permanently stops dividing but does not die. Over time, large numbers of old (or senescent) cells can build up in tissues throughout the body. These cells remain active and can release harmful substances that may cause inflammation and damage to nearby healthy cells. Senescence may play a role in the development of cancer and other diseases.


Senescent cells do not die.

Therefore, a method for treating senesent cells leads one to senolytics. For example, quercetin is a senolytic. The Neurohacker Collective manufactures a senolytic with quercetin, among other ingredients. I do not have experience with using this supplement however.


Here is a list of their ingredients and claims:

Fisetin (from Rhus succedanea Stem Extract)

Supports tissue health by helping with pruning of stressed cells*

Quercefit® Quercetin Phytosome (Sophora japonica L. Flower Extract / Phospholipid Complex from Sunflower)

Supports tissue health by helping with pruning of stressed cells*

Longvida Optimized Curcumin® Extract (from Curcuma longa Root)

Supports brain health and cognitive function*

Olive Leaf Extract

Supports management of stressed cells in joints*

Soybean Seed Extract

Supports healthy cellular functions involved in managing stressed cells*

Luteolin (from Sophora japonica L. Flower Extract)

Supports tissue health by helping with pruning of stressed cells*

Milk Thistle Seed Extract

Supports liver health and management of stressed cells*

Piperlongumine (from Piper longum Root Extract)

Supports senolytic and immune functions to help manage stressed cells*

Senactiv® (Panax notoginseng Root Extract and Rosa roxburghii Fruit Extract)

Supports exercise recovery and management of senescent cells in muscle*

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I want to thank you for all your hard work.

I'm not a paying subscriber, but I still want to make say how much I value your articles and the Friday Hope posts.

I'm not vaccinated, but I dropped out of my master program and moved to a different country.

But still many young people are vaccinated, and I fear the consequences of the mRNA and spike.

I hope with some otc supplements the damage could be limited.

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This explaines my autoimmune condition, I developed alopecia areata after my second shot of pfizer vaccine! It s been over a year and my hair is still missing from a few bald patches that i have in my beard and scalp! i wonder what damage did it make elsewere in the body! Best regards, George

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Jan 24Liked by Walter M Chesnut

“I will search for ways to treat this.” Thank you Walter ❤️

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Jan 24Liked by Walter M Chesnut

Cellular function de-evolution. If it wasn’t tragic and lethal, it would be fascinating.

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Jan 24Liked by Walter M Chesnut

So, it kind of sounds like a case of Alzheimers at the cellular level - built-in knowledge of self/function is gone. Kind of ironic when considered along with yesterday's post about this covid vaxx damage being like induced Progeria / ultra rapid aging....

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Not good news. We are unvaxxed and covid experienced (2x). Noticing immune changes. Keeping reinfection at bay with ivm and artemisia plus other supps. Possible last C19 conferred immunity but not willing to chance it again. Good news is myo has not been linked to natural infection but we really need studies differentiating between vaxxed and non.

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Thank you Walter for an easy to understand explanation for what's happening on a cellular level. Yes, I am eager to learn how long the effects last. Grateful for your sharing your findings and analyses.

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How in the world does one cope with this?

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Warning from German Health Minister!! - Immunodeficiency after COVID infection


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Jan 27Liked by Walter M Chesnut

Of possible interest and synthesis into your hypothesis. By replacing the stop codons with pseudo-uridine, what all have we wrought aside from long-lived spike protein production? What kind of monkey wrench have we thrown into the delicate intra-cellular signaling regime? At the transcriptome level?


Epub 2021 May 5.

Title: Ribosome dynamics and mRNA turnover, a complex relationship under constant cellular scrutiny


"For many years, it was believed that translation‐dependent mRNA decay was restricted to aberrant mRNAs, and that the bulk of mRNA turnover occurred mainly through a two‐step process, first with repression of translation coupled with ribosome dissociation, followed by decapping and exonucleolytic degradation of RNAs within P‐bodies (Franks & Lykke‐Andersen, 2008; Parker & Sheth, 2007). However, since the discovery of co‐translational decapping and exonucleolytic degradation, first in yeast and later in mammals, it has become very clear that translation‐dependent mRNA degradation is a major mechanism of regulation of mRNA half‐lives at the transcriptome level. From unicellular eukaryotic organisms to metazoans, the translation process appears to be constantly scrutinized by the cell, in order to trigger mRNA degradation through a myriad of pathways capable of monitoring different aspects of ribosome progression. It is now known that pathways that were initially thought to participate uniquely in mRNA quality control are also implicated in the fine tuning of a large spectrum of functional transcripts. These widespread ribosome‐dependent RNA decay mechanisms have even been harnessed by cells in order to couple translation‐dependent RNA decay with the biogenesis of functional small RNAs."

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Many thanks to IGOR (how is his cute PUPPY?) for pointing out this terrific article!

Indeed, congratulations, Wondrous Walter, for your research efforts!!

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Hello, a follower on Gettr just told me that you posted on Twitter about Prion Diseases from the jabs about a year ago. Unfortunately I am still banned there.

Could you point me to that work or reference please?


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