If say the protein encoding was flawed, due to incompleteness or fragmentation in the mRNA production, it seems that some degree of increased protein misfolding might be observed.
Might this exacerbate amyloidosis in certain individuals exposed to such conditions?
I disagree when the study says that "The molecular structure responsible for the toxicity of amyloid and amyloid intermediates is not fully understood." The action is completely understood which is why it was chosen for the bioweapon spike protein. It isn't random that seven amyloidogenic sequences distributed over the entire S protein were identified. This was designed in a lab for a very clear purpose.
The creepy/scary part about this rapid onset of cascading events that bring on sudden death is just how fast it happens. I'm convinced my young [almost 8 year old] cat who was a bit overweight [extra fat being a known co-morbidity] died this way. That evening she showed a little bit of malaise but she held on through the night until the next morning and died in her favorite chair. I had zero warning or chance to save her. The news kept saying cats have great immunity against covid. Ugh. She was exposed to a family member with covid...I so wish I'd given her Ivermectin knowing what I know now. Her slender little sister's health has been okay through it all.
I can only wonder how humans feel the night before they die so fast? Many report their loved ones being fine and then dead in their sleep without a telling symptom to alert them. It's truly horrific.
Do you suppose this explains even in the 10-20 cm long clots in the ARTERIES? I do not think so. I think amiloidosis is relevant but not enough explanations the long, dark, almost black stuff in the arterias. The color of the clots in the arterias are very different compare to the veins.
Are these similar to the amyloid plaques that interfere with the myelin sheath that hampers nerve conductivity in general depending upon stage of development i.e. MS?
So how would a Consultant test for Amyloidosis? Lets say someone I know had a heart attack a few months ago. No obvious signs as to why he had a heart attack, no clogged arteries or high cholesterol, so if they are over looking amyloidosis? It certainly put his other friends off getting boosted this Winter. He was 53. Also my daughter has heard of two peoples mothers she knows, dying suddenly in the last 3 days, both aged around 50, one had just got back off holiday, got up collapsed and died in hallway, the other was found to have died in her sleep. YIKE BLOODY YIKES.
Does anyone know if there have been any connections discussed in the literature between AL Amyloidosis and the spike protein? I know someone (a healthy male) around 40 who was diagnosed with AL Amyloidosis. They found it in a colonoscopy and fortunately it wasn't detectable in the heart region. When I started to learn last years that amyloid deposits (from the body's reaction to the spike protein) likely contributed to significant clotting and heart issues with some vaccinated individuals, I let him know and suggested he ask about if AL Amyloidosis cases have gone up significantly recently and research it some more. He has/is been treated at leading institutions like Johns Hopkins and Cleveland Clinic and he doesn't seem interested in questioning what he is being told. He has been fully vaccinated, and boosted with the Covid-19 vaccine more than once I believe, he even got boosted while on chemo for this condition which I didn't think was a good idea in case the spike protein was part of the problem. But then AL Amyloidosis has been occurring well before Covid and spike proteins so maybe there is no correlation? Anyone hear if AL Amyloidosis cases have increased, it's so rare I'd think you wouldn't need too many added cases to see the signal?
You rock Walter . 💥
Do you still consider Quercetin and Curcumin useful against amyloidosis?
If say the protein encoding was flawed, due to incompleteness or fragmentation in the mRNA production, it seems that some degree of increased protein misfolding might be observed.
Might this exacerbate amyloidosis in certain individuals exposed to such conditions?
I disagree when the study says that "The molecular structure responsible for the toxicity of amyloid and amyloid intermediates is not fully understood." The action is completely understood which is why it was chosen for the bioweapon spike protein. It isn't random that seven amyloidogenic sequences distributed over the entire S protein were identified. This was designed in a lab for a very clear purpose.
The creepy/scary part about this rapid onset of cascading events that bring on sudden death is just how fast it happens. I'm convinced my young [almost 8 year old] cat who was a bit overweight [extra fat being a known co-morbidity] died this way. That evening she showed a little bit of malaise but she held on through the night until the next morning and died in her favorite chair. I had zero warning or chance to save her. The news kept saying cats have great immunity against covid. Ugh. She was exposed to a family member with covid...I so wish I'd given her Ivermectin knowing what I know now. Her slender little sister's health has been okay through it all.
I can only wonder how humans feel the night before they die so fast? Many report their loved ones being fine and then dead in their sleep without a telling symptom to alert them. It's truly horrific.
Do you suppose this explains even in the 10-20 cm long clots in the ARTERIES? I do not think so. I think amiloidosis is relevant but not enough explanations the long, dark, almost black stuff in the arterias. The color of the clots in the arterias are very different compare to the veins.
Lisa Maria Presley!
Tweeted out to the sharpest tools I follow.
Are these similar to the amyloid plaques that interfere with the myelin sheath that hampers nerve conductivity in general depending upon stage of development i.e. MS?
Agree completely on amyloidosis but believe you are not giving due consideration to role LNP’s may be playing.
https://twitter.com/sharobarow/status/1589813895879065600?
So how would a Consultant test for Amyloidosis? Lets say someone I know had a heart attack a few months ago. No obvious signs as to why he had a heart attack, no clogged arteries or high cholesterol, so if they are over looking amyloidosis? It certainly put his other friends off getting boosted this Winter. He was 53. Also my daughter has heard of two peoples mothers she knows, dying suddenly in the last 3 days, both aged around 50, one had just got back off holiday, got up collapsed and died in hallway, the other was found to have died in her sleep. YIKE BLOODY YIKES.
https://www.frontiersin.org/articles/10.3389/fphys.2022.1081119/full?utm_source=dlvr.it&utm_medium=twitter
New Research: Paradigms of endothelial stiffening in cardiovascular disease and vascular aging
Does anyone know if there have been any connections discussed in the literature between AL Amyloidosis and the spike protein? I know someone (a healthy male) around 40 who was diagnosed with AL Amyloidosis. They found it in a colonoscopy and fortunately it wasn't detectable in the heart region. When I started to learn last years that amyloid deposits (from the body's reaction to the spike protein) likely contributed to significant clotting and heart issues with some vaccinated individuals, I let him know and suggested he ask about if AL Amyloidosis cases have gone up significantly recently and research it some more. He has/is been treated at leading institutions like Johns Hopkins and Cleveland Clinic and he doesn't seem interested in questioning what he is being told. He has been fully vaccinated, and boosted with the Covid-19 vaccine more than once I believe, he even got boosted while on chemo for this condition which I didn't think was a good idea in case the spike protein was part of the problem. But then AL Amyloidosis has been occurring well before Covid and spike proteins so maybe there is no correlation? Anyone hear if AL Amyloidosis cases have increased, it's so rare I'd think you wouldn't need too many added cases to see the signal?