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Jul 7, 2022·edited Jul 7, 2022Liked by Walter M Chesnut

Bingo Walter! I’ve been waiting for this. I measure SARS-CoV-2 Semi Quant. Total Spike antibodies in all my patients, if possible, and there appears to be a strong correlation between severity of symptoms and disease presentation and level of antibodies.

One of my patients never vaccinated receiving monthly IVIG infusions since at least early 2021 all of a sudden after her December treatment showed very elevated levels of Spike antibodies, in the many thousands where as she did not have higher than 100 previously after community acquired infection. Therefore I suspect that the IVIG supply is contaminated.

Thank you so much!!

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Nice finds!

I can help you a bit on this one. I haven't put the diagrams in articles, but have presented them during interviews---the DMED data shows a clear increase in severity of COVID-19 infections among the military (similar age/health profiles with little chance for Simpson's paradoxes). I will put this together in an article and link back here.

Cheers.

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Jul 7, 2022Liked by Walter M Chesnut

I wonder if Colostrum with high immuglobulin would be helpful for those who could not get the intravenous treatment?

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Jul 7, 2022·edited Jul 7, 2022Liked by Walter M Chesnut

ADE was always bound to happen due to the repeated use of non sterilising etc “vaccines” Thanks for this excellent article, I will share this.

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It seems to me you are saying something very important and simple. A lot depends on the neutrophil/lymphocyte ratio. In young people there is an aggressive innate response with NK cells, but as we get older the ratio turns towards greater dependence on leukocytes, causing inflammation and the neutrophils get activated because of ADE and release an enzyme that starts the amyloidosis process whilst also igniting the NLRP3 inflammasome. This combo is the essence of the pathology of SARS-2. Check your CRP and NLR blood work and take Zinc/ Quercetin /ivermectin for the virus, niacin for inflammation, and Tumeric/Lumbrokinase to dissolve amyloid and Olive leaf for all the above.

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Jul 7, 2022·edited Jul 7, 2022Liked by Walter M Chesnut

I saw that Shi Zhengli ADE paper very early on. Like, the same month it was published. It was one of the first things that convinced me to dig deeper. Very concerning.

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Jul 7, 2022Liked by Walter M Chesnut

Thanks, Walter.

I will share it to about 20 Aussie "experts", including CHOs - they are my unrequited penpals.

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Jul 7, 2022Liked by Walter M Chesnut

On the one hand we have many saying don't live in fear and go about your life and on the other hand we have Walter saying data says to limit exposures. I don't want to live in fear of exposures. We are all unvaxxed, had mild covid in Dec 2021, have good vitamin d levels and use high doses of vitamin c, probiotics and quercitin/zinc. It is difficult to know what to do. I certainly don't want more lockdowns/masks which people will want if Walter is indeed correct. Living under a rock in fear of dying is no life. I just don't believe there aren't natural ways to mitigate the harm of the spike.

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Jul 7, 2022Liked by Walter M Chesnut

Excellent, always!!!

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Would someone please describe the main gist of this post in laymen terms? This will take me a while to understand. . . a bit too technical for me.

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Jul 7, 2022Liked by Walter M Chesnut

Love the French reference of the Dreyfus Affair Walter.

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Jul 7, 2022Liked by Walter M Chesnut

Saving and sharing. Thank you Walter.

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What are the implications for someone who received monoclonal antibodies (satrovimab) back in January this year.

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Walter, every single of your posts is more amazing than even the last. I can almost hear the whistling sound of a bomb dropping every time I see your name in my feed. You are still so very missed on the twitter (as is now Jikky 🐭 x 2, sadly). I have been wondering if you think there is any utility in autophagy’s ability to clear spike or prevent the kinda of outcomes you theorize? I wonder because of a few things you and Jessica have each said over these many months and because I read a long while back a reference to the jabs lowering recipients’ ability to enter autophagy; tho I can’t say where it stuck hard with me (also considering how in the US so many are affected by metabolic dysfunction/obesity and how obesity is considered something of a dire comorbidity with the virus).

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ICU cases of Covid-19 are caused by two lethal antibodies: REGN10987 and B38. That is why Uracil was replaced with Pseudouridine, in order to sabotage the immune system in producing isomeric (binding) antibodies. Since Uracil has been substituted with Pseudouridine, this means that the vaccines are cmRNA and not mRNA: had the vaccines been mRNA, all of the vaccinees would have found themselves in ICU with severe cases of Covid-19 because of the two lethal antibodies listed above. The huge problem now is constituted by the ISOMERIC ABS.

Spike proteins = dextrorotatory prions = T-bacilli. D-prions (beta sheet prions) absorb the oxygen and cause hypoxia and micro blood clots. Spike proteins coded with Pseudouridine are even more prone to reach a dextrorotatory state (chirality).

Omicron is Mers-Cov-2 since it uses the DPP4 celullar receptor. There are no viruses only mycobacterium and mycoplasma. Sars-Cov-2 is actually M. avium accompanied by passenger mycoplasma such as Chlamydia pneumoniae. Omicron is M. influenzae (aka Mers-Cov, aka H1N1).

1915-1917 worldwide pandemic of "coronavirus", actually M. avium

1918 M. influenzae which in the course of a single day became M. africanum (galloping tuberculosis)

2019-2021 M. avium (Sars-Cov-2)

2022 M. influenzae/Omicron (Mers-Cov-2)

The difference between Omicron and Delta is that Omicron has not had, yet, its prion domain activated.

MPV is still Sars-Cov-2 (poxviridae is a mycobacterium, a variant of M. leprae), Sars-Cov-2 has some 20 sequences of amino acids from the most lethal pathogens known today (M. tuberculosis, B. anthracis, Y. pestis, M. bovis, M. leprae).

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That's disturbing. Definitely up there on the WTF scale. Table 6 in the Shi paper is not particularly comforting. Where bad is varies by tissue type and receptor type. If it happened in mers would it happen in cov2? Well that would explain some things people have been arguing about for a good part of 2021. The whole don't count the vaxed as vaxed because lots of covid in the first 2 weeks after jab then a period of likely protection (very high end antibodies) then a decline and greater susceptibility vs unvaxed. I believe that is the arch Walter is talking about. The statisticians have been saying there is ADE on the back end for a while. Looks like they are right. The avoiding of counting people as vaccinated for 14 days post jab would be an admission that people are more vulnerable during that period as well as a way to pad the numbers on effacacy so it is undoubtably worse that presented. Table 6 seems to indicate a ramp enhancement time for MERS in all but lung DPP4 but one has to wonder about the profile for tissues not tested. Makes me wonder what the covid viral and vax antibody ramp looks like for that 14 day period. Also some of the statisticians were showing negative values against control (unvaxed) meaning vax enhanced disease after a few months using the hospital/ death and cases data. If there were a paper or two profiling antibody levels by month of recovered viral and vaxed one could swag a zone where such things may well be happening based on first 2-3 weeks and maybe about 4 months to 6 months (where the booster was then recommended) on the fist 2 jabs. The signal is strong in the vax profiles which then could be compared to the viral recovered unvaxed to see if there is a problem too. However as far as I am concerned it is enough to know there is a problem overall and to think about how to manage it for prevention, if ill and post exposure of any sort.

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