Your treatment list includes some Nrf2 promoters. That is what I have found helpful and recommend. When anything inflammatory is promoting NfKB, then at the same time Nrf2 is being inhibited. The share a circadian protein so cannot both be active at the same time.
Fight back with Nrf2 promoters. Sulforaphane, resveratrol, EPA/DHA, curcumin, citrus or pomegranate peel, quercetin, genistein, others.
Yes, Glutathione precursors are the must, and all the other antioxidants will reverse this.
I have ALL people who took them alive/surviving so called Covid, including hospitalized patients in very bad condition. And I have growing number of heavy injured people to getting back on track and feeling well, but due to toxicity of those shots they must be taking antioxidants constantly.
NAC, Ivermectin- if available, Zinc, Quercetin, Vitamin C - and others: Q10, K2MK7, Vit E, Resveratrol, Vit D, Vit B, Beta Carotene, Nigella Sativa, Turmeric, etc. - they are all antioxidants and stop apoptosis of the cell.
Why Glutathione - If I would really have no money and access to anything else, I would go for N-acetylcystaine - NAC and I would add other antioxidants that available.
NAC dissolves blood clots, but also amyloid plaque. It is Glutathione precursor and glutathione is THE MASTER of all antioxidants. Basically, all other will work when Glutathione is in place.
https://madsa.org.my/how-nac-should-be-part-of-stroke-prevention-diet/ - The study by CHOP researchers suggests NAC may block the precipitation of amyloid plaque deposits, as well as help break up their formation, which could make a dramatic difference for those living with HCCAA. The study’s lead author, Dr. Hakon Hakonarson, director of CHOP’s Center for Applied Genomics, said in a news release:10
“Amyloids cannot precipitate without aggregating, so if we can prevent that aggregation with a drug [NAC] that is already available, then we could make an incredible difference in the lives of these patients.
NAC broke the oligomers into monomers (oligomers are composed of a few monomer units13), which helps prevent the formation of amyloid deposits linked to strokes.14 “Given the results from the described cellular work, it became evident that NAC could potentially be useful to treat this devastating disease,” the researchers wrote in Nature Communications.15
https://www.hindawi.com/journals/neuroscience/2019/7547382/ - It can be postulated that NAC might have reversed the effect of intraneuronal beta amyloid protein by acting on some downstream compensatory mechanisms which needs to be explored.
The antioxidant NAC has reversed the cognitive deficits and inhibited microglia activation but failed to inhibit expression of BAP positive neurons and reactive astrocytes in an animal model of AD. It can be postulated that NAC might have reversed the effect of intraneuronal beta amyloid protein by acting on some downstream compensatory mechanism which needs to be explored.
https://pubmed.ncbi.nlm.nih.gov/30641367/ How graphene affects the misfolding of human prion protein: A combined experimental and molecular dynamics simulation study
Abstract
As the broad application of graphene in the biomedical field, it is urgent and important to evaluate how the graphene affects the structure and function of the proteins in our body, especially the amyloid-related proteins. Prion protein, as a typical amyloid protein, it misfolding and aggregation will lead to serious prion diseases. To explore if graphene promotes or inhibits the formation of amyloid, here, we combined the experimental and molecular dynamics (MD) simulation methods to study the influence of graphene on the globular domain of prion protein (PrP117-231). The results from fluorescence quenching and circular dichroism spectrum showed that the addition of graphene changed the secondary structure of prion protein largely, mainly reflecting in the reduced α-helix structure and the increased coil structure, indicating graphene may strengthen the misfolding inclination of prion. To further uncover the mechanism of conformational change of prion under the induction of graphene, the all-atoms MD simulations in explicit solvent were performed. Our simulations suggest that prion protein can be quickly and tightly adsorbed onto graphene together with the weak conformational rearrangement and may reorient when approaching the surface. The Van der Waals' force drive the adsorption process. In the induction of graphene, H1 and S2-H2 loop regions of prion become unstable and prion begins to misfold partially. Our work shows that graphene can induce the misfolding of prion protein and may cause the potential risk to biosystems.
Here is the information what markers can be done to find out if oxidative stress is present: A limitation of our study, apart from the small sample size however, is that we were unable to do laboratory testing in our patients, including checking oxidative stress markers (lipid peroxides) as well as inflammatory markers (CRP, ferritin, D-dimer) and LDH which might demonstrate a change post GSH administration [[76], [77], [78]]. A randomized, controlled trial of GSH, glutathione precursors with inflammatory/oxidative stress markers should be done in the future to fully elucidate the effects of blocking NF-kappaB, and to determine the effect of GSH and antioxidants on the clinical course of COVID-19 pneumonia and ARDS. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7172740/ - Efficacy of glutathione therapy in relieving dyspnea associated with COVID-19 pneumonia: A report of 2 cases
Oxidative pathomechanisms play an important role in neurodegenerative diseases like Alzheimer's disease (AD). It has been shown that lipid peroxidation in cerebrospinal fluid (CSF) and plasma is increased in AD. To assess the role of oxidative stress in Creutzfeldt–Jakob disease (CJD), we investigated the oxidizability of lipids, the lipid composition and the levels of the antioxidants ascorbate and α-tocopherol in CSF and plasma of 15 CJD patients and 12 neurologically healthy controls. CSF and plasma lipid peroxidation was increased in CJD patients and polyunsaturated fatty acids were reduced in CSF of these patients. Ascorbate levels were lower in CSF and plasma of CJD patients, while α-tocopherol was found to be decreased in CSF but not in plasma. These results support the hypothesis that oxidative mechanisms are involved in the pathogenesis of CJD and provide a rationale for the use of antioxidants in the therapy of this disease.
Creutzfeldt-Jakob disease (CJD) " https://onlinelibrary.wiley.com/doi/abs/10.1046/j.1365-2990.2002.t01-1-00410.x " The present findings demonstrate oxidative, glycoxidative, lipoxidative and nitrative protein damage, accompanied by increased oxidative responses, in the cerebral cortex in sporadic CJD. These results provide support for the concept that oxidative stress may have important implications in the pathogenesis of prion diseases." -
So you can see all those diseases, vaccine outcomes have one common factor OXIDATIVE STRESS - but I know it, because I understand graphene toxicity mechanisms and one of the main one is oxidative stress, acute oxidative stress with all the outcomes of "Covid" and "vaccine" injury.
The toxicity of what people have been poisoned with and what's in those injections causes oxidative stress and this must be stopped and those responsible be punished.
I have a practical experience it is helping people who had the shot to survive and recover.
We're not talking about NAC in general, speculating. We are talking about a dramatic fight for lives.
But on the science side there are so many articles that prove NAC and precursors of glutathione role in prevention and treatment or sorts of diseases and acute conditions, that it would be hard to undermine those.
In conclusion, considering the present results, the prophylactic use of NAC seems to reduce the damage to the liver during radiotherapy. The results of this study indicated that the administration of NAC stimulates the anti-oxidant enzyme activities, inhibits the lipid peroxidation, and enhances the bactericidal activity of neutrophil by increasing induced MPO
activity under radiation-induced oxidative stress.
Although the findings of this study are limited to biochemical parameters, they give clues about the probability that the radioprotective effect of NAC, as shown in other studies, may be similar to that of WR ﾝ2721. In spite of WRﾝ2721, NAC, as a drug that is safe and easy to use in clinical application, has not yet been used clinically for this purpose. Further experimental studies are needed to prove this result and to rule out the potential protection of tumor cells
MPO (myeloperoxidase) is an enzyme that biodegrade graphene
Btw. Dr. Horowitz has been treating with success Lyme disease with NAC and glutathione
I would add chokeberries as antioxidant and other antioxidants
Not sure if this will be a repeat. Thought I posted a reply to you but don't see it. Anyway, very glad to see your name. Miss you on Twitter. Signed up for your sub stack, Love, Judy
Sounds like a Nuclear Disaster would be a nice way to cover the tracks... Nothing to do with another overlapping agenda of the WEF which consists in ruining Europe and controlling Eurasian heartland... #Ukraine...
We are not doomed. We can't all worry about everything. The important thing is to learn the truth, map it out, and act accordingly. This work uncovering the truth allows us to actually solve the problem. It appears that rapidly increasing numbers of our good people are becoming aware what we have a pathocracy problem. Multi-generational psychopaths have control of most levers of power. They are willing to poison and kill as many as they deem necessary in the attempt to secure their rule over us. Nuclear Disaster is not ruled out. And they are afraid. Awareness of their (formerly secret) agenda is spreading rapidly now, and every move they make to suppress the truth serves to confirm their guilt. We are in a race! Do not despair! Press on! Speak Truth!
More attention needs to be given to RAS>ROS induced pathologies and bystander effects. If it gets as far as fibrosis it's irreversible but may take years to become symptomatic. We've been warning of this for the last 2 years.
Pathogenesis and Prevention of Radiation-induced Myocardial Fibrosis
...ROS can inactivate NO and the other vascular protective agents to prevent platelet aggregation, resulting in vascular endothelial dysfunction, and then induce or aggravate the occurrence of radiation-induced myocardial fibrosis. At the same time, oxidative stress directly induces the production of inflammatory factors, which can further increase the degree of radiation-induced myocardial fibrosis. NF-κB is a protein complex that controls DNA transcription and participates in the cell’s response to stress, and it is a key link in oxidative stress and inflammatory pathways (Taunk et al., 2015).
I have never doubted this to be the case, however my view is that of a layman’s who has been researching for many years - l didn’t know whether to be relieved or absolutely terrified Walter when l read yet another excellent piece of research backed hypothesis! You are truly amazing - a huge thank you!
Sigh. The infection shortens lives too. The omicrons have the Prp prion and amyloid promoting sequences silenced. I've had Wuhan and 2 omicrons. I've come to the conclusion based in research and the doctors (and Walter's work too!) that given the spike hangs out in reservoirs even after successfully getting over the infections, it is a big insult to the immune system (HIVs, Murine Malaria, TSS, Staph A, TB, prion, amyloid etc promoting sequences, and so much more, optimized for Humans). Also: https://www.thailandmedical.news/news/breaking-hypothesis-that-majority-exposed-to-sars-cov-2-will-have-shortened-lifespans-validated-by-study-showing-nsp2-impairs-human-4ehp-gigyf2-comple I have a follow on I must find that established this is the case, also Bart Classon predicted this.
Would we find a parallel to encephalitis lethargica, which was potentially a sequela to Spanish Flu? Parkinsonism. Could this be a resurgence of “sleeping sickness”? I see it beginning to appear now.
Given the long-term damage this virus seems to be now causing, what is your view on people facing multiple re-infections going on long-term opiod therapy as a prophylaxis against infection? The evidence seems to be overwhelming that people on long-term opiod therapy (morphine, methadone, oxycodone etc) are not getting infected:
I'm wondering why COVID patterns have a strong correlation with the expansion of cell towers/satellites and the damage clinically mirrors it, yet most people think there is no relationship to radiation. Are we just doing the gain-of-function dance until we die?
Walter - why don’t you think the spike protein is generating electrostatic forces and radiating a field in the ionising energy range, creating oxidative stress and free radicals?
Your treatment list includes some Nrf2 promoters. That is what I have found helpful and recommend. When anything inflammatory is promoting NfKB, then at the same time Nrf2 is being inhibited. The share a circadian protein so cannot both be active at the same time.
Fight back with Nrf2 promoters. Sulforaphane, resveratrol, EPA/DHA, curcumin, citrus or pomegranate peel, quercetin, genistein, others.
Thank you, always, Jennifer. Excellent.
Very welcome, Walter.
It was the topic of my first post written on Substack - Nrf2 and NfKb: 2 proteins to know. https://denutrients.substack.com/p/nrf2-and-nf-kb-2-proteins-to-know
Yes, Glutathione precursors are the must, and all the other antioxidants will reverse this.
I have ALL people who took them alive/surviving so called Covid, including hospitalized patients in very bad condition. And I have growing number of heavy injured people to getting back on track and feeling well, but due to toxicity of those shots they must be taking antioxidants constantly.
NAC, Ivermectin- if available, Zinc, Quercetin, Vitamin C - and others: Q10, K2MK7, Vit E, Resveratrol, Vit D, Vit B, Beta Carotene, Nigella Sativa, Turmeric, etc. - they are all antioxidants and stop apoptosis of the cell.
Why Glutathione - If I would really have no money and access to anything else, I would go for N-acetylcystaine - NAC and I would add other antioxidants that available.
NAC dissolves blood clots, but also amyloid plaque. It is Glutathione precursor and glutathione is THE MASTER of all antioxidants. Basically, all other will work when Glutathione is in place.
https://madsa.org.my/how-nac-should-be-part-of-stroke-prevention-diet/ - The study by CHOP researchers suggests NAC may block the precipitation of amyloid plaque deposits, as well as help break up their formation, which could make a dramatic difference for those living with HCCAA. The study’s lead author, Dr. Hakon Hakonarson, director of CHOP’s Center for Applied Genomics, said in a news release:10
“Amyloids cannot precipitate without aggregating, so if we can prevent that aggregation with a drug [NAC] that is already available, then we could make an incredible difference in the lives of these patients.
NAC broke the oligomers into monomers (oligomers are composed of a few monomer units13), which helps prevent the formation of amyloid deposits linked to strokes.14 “Given the results from the described cellular work, it became evident that NAC could potentially be useful to treat this devastating disease,” the researchers wrote in Nature Communications.15
https://www.hindawi.com/journals/neuroscience/2019/7547382/ - It can be postulated that NAC might have reversed the effect of intraneuronal beta amyloid protein by acting on some downstream compensatory mechanisms which needs to be explored.
The antioxidant NAC has reversed the cognitive deficits and inhibited microglia activation but failed to inhibit expression of BAP positive neurons and reactive astrocytes in an animal model of AD. It can be postulated that NAC might have reversed the effect of intraneuronal beta amyloid protein by acting on some downstream compensatory mechanism which needs to be explored.
https://pubmed.ncbi.nlm.nih.gov/30641367/ How graphene affects the misfolding of human prion protein: A combined experimental and molecular dynamics simulation study
Abstract
As the broad application of graphene in the biomedical field, it is urgent and important to evaluate how the graphene affects the structure and function of the proteins in our body, especially the amyloid-related proteins. Prion protein, as a typical amyloid protein, it misfolding and aggregation will lead to serious prion diseases. To explore if graphene promotes or inhibits the formation of amyloid, here, we combined the experimental and molecular dynamics (MD) simulation methods to study the influence of graphene on the globular domain of prion protein (PrP117-231). The results from fluorescence quenching and circular dichroism spectrum showed that the addition of graphene changed the secondary structure of prion protein largely, mainly reflecting in the reduced α-helix structure and the increased coil structure, indicating graphene may strengthen the misfolding inclination of prion. To further uncover the mechanism of conformational change of prion under the induction of graphene, the all-atoms MD simulations in explicit solvent were performed. Our simulations suggest that prion protein can be quickly and tightly adsorbed onto graphene together with the weak conformational rearrangement and may reorient when approaching the surface. The Van der Waals' force drive the adsorption process. In the induction of graphene, H1 and S2-H2 loop regions of prion become unstable and prion begins to misfold partially. Our work shows that graphene can induce the misfolding of prion protein and may cause the potential risk to biosystems.
https://www.ahajournals.org/doi/10.1161/CIRCULATIONAHA.117.027290
Potent Thrombolytic Effect of N-Acetylcysteine on Arterial Thrombi
I really highly recommend to look at this: https://anamihalceamdphd.substack.com/p/computerized-thermographic-imaging?utm_source=substack&utm_medium=email - https://rumble.com/v1gzhzh-computerized-thermographic-imaging-and-live-blood-analysis-post-c19-injecti.html
This is important as it lists many antioxidants and Myeloperoxidase biodegrades graphene https://www.frontiersin.org/articles/10.3389/fphys.2020.00433/full - Medicinal Herbal Compounds With the MPO-Inhibiting Activity Showing Antioxidant, Anti-Inflammation, and Neuroprotective Effects - https://www.frontiersin.org/files/Articles/528444/fphys-11-00433-HTML/image_m/fphys-11-00433-t002.jpg - Table 2. Natural compound inhibiting MPO for protecting ischemic stroke injury.
Here is the information what markers can be done to find out if oxidative stress is present: A limitation of our study, apart from the small sample size however, is that we were unable to do laboratory testing in our patients, including checking oxidative stress markers (lipid peroxides) as well as inflammatory markers (CRP, ferritin, D-dimer) and LDH which might demonstrate a change post GSH administration [[76], [77], [78]]. A randomized, controlled trial of GSH, glutathione precursors with inflammatory/oxidative stress markers should be done in the future to fully elucidate the effects of blocking NF-kappaB, and to determine the effect of GSH and antioxidants on the clinical course of COVID-19 pneumonia and ARDS. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7172740/ - Efficacy of glutathione therapy in relieving dyspnea associated with COVID-19 pneumonia: A report of 2 cases
The Role of Oxidative Stress in Neurodegenerative Diseases - PMC (nih.gov) / https://pubmed.ncbi.nlm.nih.gov/26713080/
"Abstract
Oxidative pathomechanisms play an important role in neurodegenerative diseases like Alzheimer's disease (AD). It has been shown that lipid peroxidation in cerebrospinal fluid (CSF) and plasma is increased in AD. To assess the role of oxidative stress in Creutzfeldt–Jakob disease (CJD), we investigated the oxidizability of lipids, the lipid composition and the levels of the antioxidants ascorbate and α-tocopherol in CSF and plasma of 15 CJD patients and 12 neurologically healthy controls. CSF and plasma lipid peroxidation was increased in CJD patients and polyunsaturated fatty acids were reduced in CSF of these patients. Ascorbate levels were lower in CSF and plasma of CJD patients, while α-tocopherol was found to be decreased in CSF but not in plasma. These results support the hypothesis that oxidative mechanisms are involved in the pathogenesis of CJD and provide a rationale for the use of antioxidants in the therapy of this disease.
Creutzfeldt-Jakob disease (CJD) " https://onlinelibrary.wiley.com/doi/abs/10.1046/j.1365-2990.2002.t01-1-00410.x " The present findings demonstrate oxidative, glycoxidative, lipoxidative and nitrative protein damage, accompanied by increased oxidative responses, in the cerebral cortex in sporadic CJD. These results provide support for the concept that oxidative stress may have important implications in the pathogenesis of prion diseases." -
So you can see all those diseases, vaccine outcomes have one common factor OXIDATIVE STRESS - but I know it, because I understand graphene toxicity mechanisms and one of the main one is oxidative stress, acute oxidative stress with all the outcomes of "Covid" and "vaccine" injury.
Btw., Azithromycin has antioxidant properties - this is why it works so well for Covid: https://rosacea-support.org/azithromycin-zithromax-is-anti-oxidant.html and https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4368079/ - same for Doxycycline.
The toxicity of what people have been poisoned with and what's in those injections causes oxidative stress and this must be stopped and those responsible be punished.
What do you think about this-just saw it on another Substack. Oy. (Edit: actually it’s from a commenter here above)
https://www.science.org/content/blog-post/n-acetyl-cysteine-warning-shot
I purposedly included an article on gamma radiation and oxidative stress...
Another MUST read and listen. My research comes to pretty much same conclusions: https://expose-news.com/2022/08/29/covid-injections-contain-hydrogel-and-graphene-oxide/ and https://rumble.com/v1h9piz-the-jab-its-poison-and-will-kill-you.html
I have a practical experience it is helping people who had the shot to survive and recover.
We're not talking about NAC in general, speculating. We are talking about a dramatic fight for lives.
But on the science side there are so many articles that prove NAC and precursors of glutathione role in prevention and treatment or sorts of diseases and acute conditions, that it would be hard to undermine those.
Pay attention this:
https://citeseerx.ist.psu.edu/viewdoc/download?doi=10.1.1.1050.1705&rep=rep1&type=pdf
In conclusion, considering the present results, the prophylactic use of NAC seems to reduce the damage to the liver during radiotherapy. The results of this study indicated that the administration of NAC stimulates the anti-oxidant enzyme activities, inhibits the lipid peroxidation, and enhances the bactericidal activity of neutrophil by increasing induced MPO
activity under radiation-induced oxidative stress.
Although the findings of this study are limited to biochemical parameters, they give clues about the probability that the radioprotective effect of NAC, as shown in other studies, may be similar to that of WR ﾝ2721. In spite of WRﾝ2721, NAC, as a drug that is safe and easy to use in clinical application, has not yet been used clinically for this purpose. Further experimental studies are needed to prove this result and to rule out the potential protection of tumor cells
MPO (myeloperoxidase) is an enzyme that biodegrade graphene
Btw. Dr. Horowitz has been treating with success Lyme disease with NAC and glutathione
I would add chokeberries as antioxidant and other antioxidants
Not sure if this will be a repeat. Thought I posted a reply to you but don't see it. Anyway, very glad to see your name. Miss you on Twitter. Signed up for your sub stack, Love, Judy
Thanks Judy / Elspeth. Nice to be able to reconnect!
PS: I'm Elspeth on sub stack
Sounds like a Nuclear Disaster would be a nice way to cover the tracks... Nothing to do with another overlapping agenda of the WEF which consists in ruining Europe and controlling Eurasian heartland... #Ukraine...
We are so doomed...
Let's hope not. But yeah, the nuclear thing is worrying, for sure.
I miss your Twitter :(
Thanks for all the good work ! Best
Adrien
"the nuclear thing is worrying"
... yet we act is if that is not a thing...
We are not doomed. We can't all worry about everything. The important thing is to learn the truth, map it out, and act accordingly. This work uncovering the truth allows us to actually solve the problem. It appears that rapidly increasing numbers of our good people are becoming aware what we have a pathocracy problem. Multi-generational psychopaths have control of most levers of power. They are willing to poison and kill as many as they deem necessary in the attempt to secure their rule over us. Nuclear Disaster is not ruled out. And they are afraid. Awareness of their (formerly secret) agenda is spreading rapidly now, and every move they make to suppress the truth serves to confirm their guilt. We are in a race! Do not despair! Press on! Speak Truth!
You should also read about heavy metal toxicity symptoms and treatment. Lots of things that make you go hmm in those lists.
Thanks for your diligent and persistent work. I just upgraded to a paid subscription.
Thank you for your appreciation and support. I will keep working.
More attention needs to be given to RAS>ROS induced pathologies and bystander effects. If it gets as far as fibrosis it's irreversible but may take years to become symptomatic. We've been warning of this for the last 2 years.
Pathogenesis and Prevention of Radiation-induced Myocardial Fibrosis
...ROS can inactivate NO and the other vascular protective agents to prevent platelet aggregation, resulting in vascular endothelial dysfunction, and then induce or aggravate the occurrence of radiation-induced myocardial fibrosis. At the same time, oxidative stress directly induces the production of inflammatory factors, which can further increase the degree of radiation-induced myocardial fibrosis. NF-κB is a protein complex that controls DNA transcription and participates in the cell’s response to stress, and it is a key link in oxidative stress and inflammatory pathways (Taunk et al., 2015).
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5464468/
Pomegranate and high dose niacin plus melatonin, can be very helpful.
Review article on Pomegranate and ROS -
via SIRT3 effects https://www.hindawi.com/journals/omcl/2016/2927131/
As many polyphenols as you can get. Resveratrol is another great source.
Pyconegol
https://www.webmd.com/diet/health-benefits-pycnogenol#1
(Surprised pleasantly to see it on WebMD)
Thanks, noted!
I have never doubted this to be the case, however my view is that of a layman’s who has been researching for many years - l didn’t know whether to be relieved or absolutely terrified Walter when l read yet another excellent piece of research backed hypothesis! You are truly amazing - a huge thank you!
Thank you once again and an additional thank you for pointing us to some potentail ways to fight back.
Is this with vax or original Covid infection or both?
This is an excellent question! One perhaps Walter can inform us about - l certainly would like to know as a “non “axed” ..
Sigh. The infection shortens lives too. The omicrons have the Prp prion and amyloid promoting sequences silenced. I've had Wuhan and 2 omicrons. I've come to the conclusion based in research and the doctors (and Walter's work too!) that given the spike hangs out in reservoirs even after successfully getting over the infections, it is a big insult to the immune system (HIVs, Murine Malaria, TSS, Staph A, TB, prion, amyloid etc promoting sequences, and so much more, optimized for Humans). Also: https://www.thailandmedical.news/news/breaking-hypothesis-that-majority-exposed-to-sars-cov-2-will-have-shortened-lifespans-validated-by-study-showing-nsp2-impairs-human-4ehp-gigyf2-comple I have a follow on I must find that established this is the case, also Bart Classon predicted this.
Would we find a parallel to encephalitis lethargica, which was potentially a sequela to Spanish Flu? Parkinsonism. Could this be a resurgence of “sleeping sickness”? I see it beginning to appear now.
I thought of your prescient work when I read the headline of this article: https://newspunch.com/hundreds-of-children-diagnosed-with-childhood-dementia-doctors-baffled/
Thank you for your tireless work.
How sad
Doxycycline, eapecially along vitamin C seems to lower TLR-2 and other inflammatory cytokines
https://www.researchgate.net/publication/342738073_Clinical_and_Immunological_Response_to_Doxycycline_Versus_Doxycycline_Plus_Vitamin_C_in_Patients_with_Acne_Vulgaris#pf3
It also reduces nf-kb
There's an in vitro study that suggests that low doses without antibiotic activity still reduce inflammatory cytokines.
Given the long-term damage this virus seems to be now causing, what is your view on people facing multiple re-infections going on long-term opiod therapy as a prophylaxis against infection? The evidence seems to be overwhelming that people on long-term opiod therapy (morphine, methadone, oxycodone etc) are not getting infected:
https://www.bjanaesthesia.org.uk/article/S0007-0912(21)00553-5/pdf
Interesting. What about weed?
Dear Mr. Chesnut, when you talk about "spike protein", you mean the "harmless piece" encoded in the hackcines - right?
There are many variants with many spikes :)... which is NOT exactly the "spike protein"...
I'm wondering why COVID patterns have a strong correlation with the expansion of cell towers/satellites and the damage clinically mirrors it, yet most people think there is no relationship to radiation. Are we just doing the gain-of-function dance until we die?
Can you provide links to this information?
There are clues everywhere, but here are some places to start:
https://www.europarl.europa.eu/RegData/etudes/BRIE/2020/646172/EPRS_BRI(2020)646172_EN.pdf
https://betterness.com/wp-content/uploads/TheInvisibleRainbow.pdf
http://www.xinhuanet.com/english/2019-10/31/c_138517734.htm
https://emrabc.ca/?p=17235
https://en.wikipedia.org/wiki/List_of_Starlink_launches
https://emfmadness.wordpress.com/2020/04/28/is-there-an-association-between-covid-19-cases-deaths-and-5g-in-the-united-states/
This is the tip of the iceberg. It's baffling to me how so many ignore it or call it crazy.
Walter - why don’t you think the spike protein is generating electrostatic forces and radiating a field in the ionising energy range, creating oxidative stress and free radicals?
The graphene oxide may be providing that.
Shocking: https://anamihalceamdphd.substack.com/p/computerized-thermographic-imaging?utm_source=substack&utm_medium=email - https://rumble.com/v1gzhzh-computerized-thermographic-imaging-and-live-blood-analysis-post-c19-injecti.html