Persistent elevation of these cytokines in particular, as the study found in PASC, correlate beyond reasonable doubt with pro-tumor pathways, atherosclerosis & autoimmunity. I dedicated a whole Substack to Il-6 and look forward to seeing your full write up. I guess we should mention persistence (+ accumulation?) in the ovaries too and the implications for that.

IL-6 Activities in the Tumour Microenvironment. Part 1

"The predominant role of IL-6 in cancer is the promotion of tumour growth. The interaction of IL-6 and its receptor-activated JAKs with following induction/activation of STAT3 through tyrosine phosphorylation and subsequent transcription of target genes [9] is vital in cancer formation."


Spike protein (inc vax) induced immunodeficiency & carcinogenesis megathread #26: Interleukin-6 (IL-6) induced pathology



Interleukin 8 and cardiovascular disease

"Interleukin (IL)-8 was first characterized in 1987. Since then, knowledge regarding its role in leucocyte trafficking and activation has advanced rapidly, especially in the field of cardiovascular disease. In the scientific literature, there is sufficient evidence to support beyond any doubt the involvement of IL-8 in the establishment and preservation of the inflammatory micro-environment of the insulted vascular wall."


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"persistence (+ accumulation?) in the ovaries too and the implications " I am assuming that these females will not only be non-fertile, they will start displaying symptoms of primary ovarian failure if of an age to do so; early onset menopause. If too young, there may be failure to sexually maturate due to lack of hormones; there would be cascading effects due to low estrogen. Of course, the effects on their immune systems would be as/or more important.

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A truly horrifying scenario.

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I'm just a layman, but the Results and Conclusions sections of the paper abstract sound very ominous to me.

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Jul 16, 2022Liked by Walter M Chesnut

thank you for all your hard work and dedication.

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I always smile when you say you have been proven right because I don't know of anyone who even considers the possibility that you may be wrong anymore!

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UVB also generates cytokines such as interleukin-8 (IL-8) in a PAFR-dependent manner. Which in turn can generate microvesicle particles, as part of the stress response.


This would explain why we are seeing so many cases of Covid now in the middle of summer, when pre vaccine rollout we did not.

The environmental UVB is now acting as the stressor and “energising” the spike protein and inhibiting the immune response.

Moreover, UVB phototherapy is associated with lower classical monocyte frequencies compared to controls and higher frequencies of intermediate monocytes, but only during the phototherapy treatment period.


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Do you guys believe that the jabs might be causing the immune system (possibly the intracellular immune system) to cleave the spike proteins from virus reinfections in different ways, as compared to unjabbed people?

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Jul 16, 2022·edited Jul 16, 2022

Recombinant Omicron (Mers-Cov-2)/Delta (Sars-Cov-2) variant: BA.5/AY.45


If Omicron is starting to activate its prion domain, there might be a repeat of the 1918 scenario, this time with a much stronger catalyst.

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The paper tells about findings reported by Bruce Patterson several months ago in an interview.unfor Fig 4 and 5 are missing in the version that was downloaded just now.

One commenter noticed -from own experience-that the injections seem to enhance symptoms after infection-Last name of Bruce is missing in the title.Sloppy editing but bombshell

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And there is a need to discuss pure blood long covid (per different variants), acute vax injuries, LC like symptoms after vax, LC then vax, vax then LC, etc differently. All of these scenarios are vastly different, yet they pretend all this is LC.

This is a disaster. We must begin discussing various scenarios in different ways.

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PASC involves cofactor deficiencies (eg. coenzyme deficiencies such as vit B1, B2, B12, D at least and likely way more. Hint: hair loss is not a stress response). May not show up on blood tests. I think we have a DM2 like situation.

spike > fibrin amyloid microclots > microischemia > waste congestion > reactions have a hard time accessing cofactors > decreased cellular respiration > gradual, diffuse multi organ mal functions.

and this is just one possibility. There are a lot of things going on simultaneously.

Don't even get me started on vax injuries.

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Someone just asked if you can get spike proteins from a vaxxed person after sex. I think it is feasible. I think the real question is, can you get mRNA from a vaxxed person? The scary thing is that the mRNA recreates the spike proteins over and over. What do you think?

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RNA, especially N-1 methylpseudouridine substituted RNA can generate many times its weight in mutant spike proteins.

That's why the graphene theory is nonsense as jabs only contain very little graphene and it doesn't snowball into large amounts of proteins like RNA doea.

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Is there any credible evidence that the jabs contain graphene?

If that's the case, why aren't they publishing in journals?

There are so many journals available that there should be no excuse.

I am having a hard time understanding the researchers.

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Would love to hear your thoughts on this Walter. I remember that you have written extensively about this. Now they’re saying that the same technology can fix it.


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I thought the spike in the vaccines had proline substitutions to lock it into prefusion configuration. If this is correct, how can S1 be cleaved?

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I am sorry this study is very very unconvincing.

The conclusion is very conjectural.

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Explain why it's unconvincing?

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will answer more in details over the weekend.

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