FURTHER EVIDENCE PROVING MY HYPOTHESIS THAT THE CLEAVED S1 UNIT CAUSES COVID/PASC (SYMPTOMS?) POST mRNA COVID VACCINES
Published July 12, 2022
I hope to have my unifying “Horsemen” hypothesis completed this weekend.
Meanwhile, I wish to bring to everyone’s attention that I was indeed, again unfortunately, correct when I hypothesized that the cleaved S1 unit from mRNA COVID vaccines induces COVD/PASC SYMPTOMS (?) (I question leaving it at “symptoms”).
SARS-CoV-2 S1 Protein Persistence in SARS-CoV-2 Negative Post-Vaccination Individuals with Long COVID/ PASC-Like Symptoms
https://www.researchsquare.com/article/rs-1844677/v1
Persistent elevation of these cytokines in particular, as the study found in PASC, correlate beyond reasonable doubt with pro-tumor pathways, atherosclerosis & autoimmunity. I dedicated a whole Substack to Il-6 and look forward to seeing your full write up. I guess we should mention persistence (+ accumulation?) in the ovaries too and the implications for that.
IL-6 Activities in the Tumour Microenvironment. Part 1
"The predominant role of IL-6 in cancer is the promotion of tumour growth. The interaction of IL-6 and its receptor-activated JAKs with following induction/activation of STAT3 through tyrosine phosphorylation and subsequent transcription of target genes [9] is vital in cancer formation."
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6765074/#:~:text=The%20predominant%20role%20of%20IL%2D6%20in%20cancer%20is%20the,is%20vital%20in%20cancer%20formation.
Spike protein (inc vax) induced immunodeficiency & carcinogenesis megathread #26: Interleukin-6 (IL-6) induced pathology
https://doorlesscarp953.substack.com/p/spike-protein-inc-vax-induced-immunodeficiency-10d
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Interleukin 8 and cardiovascular disease
"Interleukin (IL)-8 was first characterized in 1987. Since then, knowledge regarding its role in leucocyte trafficking and activation has advanced rapidly, especially in the field of cardiovascular disease. In the scientific literature, there is sufficient evidence to support beyond any doubt the involvement of IL-8 in the establishment and preservation of the inflammatory micro-environment of the insulted vascular wall."
https://academic.oup.com/cardiovascres/article/84/3/353/455195
I'm just a layman, but the Results and Conclusions sections of the paper abstract sound very ominous to me.