My research took us to the same place.

microRNA - Drosha & Dicer.

"Mammalian genomes are pervasively transcribed with most transcripts apparently not associated with coding functions An increasing number of ncRNAs have been shown to play a variety of relevant cellular functions, often with estimated very low expression levels DICER and DROSHA are two RNase type III enzymes that process ncRNAs hairpin structures to generate small double-stranded RNAs" - https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3442236/

GP-120 "HIV-1 actively suppresses expression of the polycistronic miRNA cluster, miR-17/92, an inhibition of the miRNA pathway that is required for efficient viral replication (Triboulet et al., 2007). Instead of altering host miRNA expression, viruses could use an alternative mechanism involving miRNA-mediated host-pathogen interactions" - https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2763548/

"Small RNAs, such as microRNAs (miRNAs), are fundamental regulators of host gene expression programs, including antiviral innate immunity genes...This analysis revealed that the small RNA has precise 5′ and 3′ ends characteristic of a bonafide miRNA, suggesting that it is not a degradation product..we show the presence of a stem–loop structure that is processed by DICER1-mediated cleavage in vitro. Third, RNAi experiments to deplete human Dicer during SARS-CoV-2 infection also show reduced accumulation of the viral miRNA and human miRNAs" - we show the presence of a stem–loop structure that is processed by DICER1-mediated cleavage in vitro. Third, RNAi experiments to deplete human Dicer during SARS-CoV-2 infection also show reduced accumulation of the viral miRNA and human miRNAs

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"Mature miRNAs are produced from primary transcripts (pri-miRNAs), which are processed by Drosha proteins into precursor miRNAs (pre-miRNAs), each of which consists of 5p and 3p arms and a terminal loop. The pre-miRNAs are transported to the cytoplasm by exportin 5 and are divided by Dicer to release the terminal loop and 5p/3p duplex.

"The downregulation of these miRNAs in patients with COVID-19 leads to IL-6/IL-6R hyperactivation by directly targeting the 3'UTR of IL-6/IL-6R, thereby enhancing the cytokine storm induced by SARS-CoV-2 infection

"Hsa-miR-18 and hsa-miR-125b play central roles in acute renal injury in patients with SARS-CoV-2 infections by directly binding to ACE2.

"Accumulating evidence indicates that ceRNA networks link the functions of protein-coding mRNAs with those of noncoding RNAs such as miRNAs, long noncoding RNAs 52, pseudogenic RNAs, and circular RNAs 51, thereby affecting and regulating the expression of target genes. Because host miRNAs can bind to the coding DNA sequence (CDS) regions of viral RNAs, even without interfering with viral RNA function, overconsumption of host miRNAs (known as the sponge effect) may lead to a reduction in the availability of such miRNAs

"miRNAs are involved in sensory system development, oxidative responses, autophagy, lung development, stress-activated MAPK signaling, and oxidative stress-induced neuron death. A previous study reported that patients with SARS-CoV-2 infection have excessive reactive oxygen species (ROS) levels, which facilitates the cascade of biological events that drive pathological host responses

Fulvic Acids & Polyphenols - the fix. Been saying this, on a cellular level since day 1. "Natural compounds can block SARS-CoV-2 infection by regulating miRNA expression - Polyphenols are the most abundant dietary antioxidants and are commonly found in fruits, vegetables, chocolate and wine"

- https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9346380/

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VAIDS. causes SPED on a miRNA level. "whereas serum TNF-α levels were associated with systemic adverse reactions and with specific antibody titers. Interestingly, EV miR-92a-2-5p levels in sera were negatively correlated with degrees of adverse reactions, and EV miR-148a levels were associated with specific antibody titers. Our data suggest a potential of circulating EV miRNAs as biomarkers for vaccine efficacy and adverse

"Our study showed a correlation of specific antibody titers with serum TNF-α levels after the second dose. TNF-α has been reported to be required for the formation of primary B-cell follicles and essential for the production of antigen-specific IgG32. Recombinant TNF-α can also promote B-cell proliferation33. TNF-α is secreted from myeloid cells and B- and T cells in response to antigens34. Hence, TNF-α is expected to be secreted from lymphocytes in response to vaccination, resulting in augmented production of specific antibodies against the viral spike protein.

"EVs include exosomes, microvesicles (also called ectosomes), and apoptotic bodies.

- https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9346380/

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GP-120 - a prion

"HIV-1 can reshape the miRNA expression profile of infected cells (21). One interpretation of this latter finding is that the virus has learned to repress the expression of virus-targeted miRNAs, while enhancing the expression of propitious miRNAs that up-regulate protein factors that benefit HIV-1 replication

"HIV-1 was shown to affect the levels of several miRNAs to change their expression profile (30). Tat protein was also shown to deregulate expression levels of selected miRNAs, including the neuronal mir-128, in primary cortical neurons

"Interestingly, some targets of the up-regulated miRNAs have been associated with neurological diseases. For instance, miR-378 has been shown to target the CYP2E1 gene, a cytochrome p450 isoform whose polymorphism is associated with Parkinson disease (32) and is found tightly associated with dopamine-containing cells in the substantia nigra (33). Accordingly, we observe a 2-fold decrease in the expression of CYP2E1 in neurons treated with Tat in addition to an increase in miR-1 expression.

"miRNAs were also shown to be involved in the pathogenesis of neuroblastoma and astrocytoma

"Among known miR-34a targets, p53 and SIRT1 genes were the most studied and shown to be involved in apoptosis or cell survival (59). miR-34a was recently shown to behave like a tumor suppressor in brain tumors and glioma stem cells (74). In addition to its involvement in tumors and neurodegenerative diseases, miR-34a was also shown to play a role in psychiatric problems.

"However, it is well described that HIV-1 benefits from deregulating the miRNAs of the host cell (26). This theory is supported by the fact that miRNA-processing enzymes Drosha and Dicer are silenced to reduce generation in the cell of mature miRNAs (Ref. 26 and references within). This phenomenon can lead to a robust HIV-1 replication and preventing some miRNAs (e.g. miR-150 or miR-223) from silencing, reducing, or even delaying viral replication

- https://www.sciencedirect.com/science/article/pii/S0021925820504614

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Merry Christmas and happy new year. This convo is above my pay grade

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Many thanks for your much needed pro bono service.

Have a happy Christmas and best for 2023!

We need you to be match-fit for 2023 :)

Sydney, Australia

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Have you watched Rounding the Earth's video with Marc Girardot, Jessica Rose, Matthew Crawford, JJ Couey, and Liam Sturgess? Marc's bolus theory is gaining ground with the other experts and they are moving away from blaming spike for the adverse events. I am getting more and more confused. Some say covid, some the spike protein, some the LNP's and now Marc says it is from poor injection protocols.

I am unjabbed but am still having adverse effects from my first covid infection 2 months ago.

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I think Marc's bolus theory makes a lot of sense. I also think there are damage mechanisms related to the spike protein. I don't understand why it would be difficult to consider that both bolus and spike might be viable damage mechanisms.

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I think there is some merit in the bolus theory, but I feel Walter is right over the target of the bioweapon shots. The campaign is being run under EAU by the DOD and HHS. See:




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Tumeric, vitamin D, antiinflamation diet. Vit C etc...

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Even when I don’t understand it all, I read every word you write. I’m just so happy to know you are continuing to tackle this beast. Your contributions are valued. Thank you for working so diligently!

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hi walter - thought it might be of interest. people in the know are starting to talk ... this guy is the president of the international vascular society and raises concerns about covid vaccines in relation to cardiovascular problems. my guess he is probably scared about himself and his family as well, most likely all vaxxed.


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Take a look at the presentation Cancer as a Trophoblastic Disease by Jeffrey Dach.


I have every reason to believe that everybody reading your latest articles will get quite a few things to ponder there.

I believe that dipyridamole could prove to be a very cheap and effective anti-spike and anticancer drug.

That´s why it´s not used.

Dipyridamole prevents platelet activation, downregulates TGFβ1, VEGF, PDGF, Wnt, MAPK/ERK, NF-kB, infiltration of tumor associated macrophages and so on.


Other great source for info about off label cancer treatments is the page by Daniel Stanciu here:


Merry Christmas and Happy New Year!

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.....soooo, would I be OVERsimplifying the 'math' to SOLVE cancer as, 'gp120-provoked clots + fibrin (i.e., mutated and / or excess collagen - with altered galvanic potentialities?) = tumors', Walter?

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Interesting tweet from Justin Hart that seems relevant to the research WMC Research is doing.


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Walter come back to twitter please, you are a valuable contributor to the truth. You need a wider audience.

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Just became a paid member.

Keep up the good work!

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