CPR does not resuscitate to begin with. At best it delays ischemia and thus extends the window for electrical defibrillation leading to full or near-full recovery. But the defibrillation does the work, and that is only for VF/VT arrest. If it's asystole or PEA then CPR is just performative, doesn't improve recovery.
WHY YOUNG, HEALTHY ATHLETES ARE NOT BEING RESUSCITATED. THE MICROVASCULATURE HAS BEEN DESTROYED.
CPR does not resuscitate to begin with. At best it delays ischemia and thus extends the window for electrical defibrillation leading to full or near-full recovery. But the defibrillation does the work, and that is only for VF/VT arrest. If it's asystole or PEA then CPR is just performative, doesn't improve recovery.
So non-resuscitation just means non-VF/VT.
Thank you!
If you did not watch it yet, kindly consider watching Jessica Rose and Wondrous Walter on gigaohmbiological on Twitch.
https://m.twitch.tv/gigaohmbiological
A fantastic exchange of information! Wondrous Walter was, of course, WONDROUS!
Might this microvascular damage also explain the sudden increase in preeclampsia? https://www.newyorker.com/science/annals-of-medicine/why-a-life-threatening-pregnancy-complication-is-on-the-rise
Yep. Can’t restart the heart if the veins and/or capillaries are shot.
as always, many thanks for your relentless pursuit of truth!!
another tennis player with sudden health issues stopping in the middle of the game.
https://www.youtube.com/watch?v=uYEcH1xrH0A&t=3s
I believe that the sp!ke pr0te!n might be impairing local thyroid hormone metabolism in the vascular endothelia.
The same impairment happens in obesity:
https://www.endocrine-abstracts.org/ea/0073/ea0073aep327
And triiodothyronine (T3 thyroid hormone) seems to reduce endothelial damage following myocardial infarction:
https://www.ahajournals.org/doi/10.1161/hyp.70.suppl_1.p247