THE SPIKE PROTEIN’S EFFECT ON THE MICROVASCULATURE OF THE HEART AND OTHER ORGANS IS EQUIVALENT TO A SILENT “HEART ATTACK.” ADDITIONAL DAMAGE FROM ISCHEMIA-REPERFUSION INJURY.
THE SPIKE PROTEIN’S EFFECT ON THE MICROVASCULATURE OF THE HEART AND OTHER ORGANS IS EQUIVALENT TO A SILENT “HEART ATTACK.” ADDITIONAL DAMAGE FROM ISCHEMIA-REPERFUSION INJURY.
Is all Spike Protein damage based upon individual’s level of Microvascular Occlusion? And, does each exposure inexorably increase an individual’s Microvascular Occlusion?
It is very clear that the microvascular damage caused by the Spike Protein’s attack on the endothelium, in essence, replicates a “mild” heart attack. By virtue of its effect on the microvasculature, it also induces “liver attacks,” “brain attacks,” “kidney attacks” and so forth via the same mechanism. Also, the post-myocardial infarction damage of ischemia-reperfusion injury, additionally, occurs.
SARS-CoV-2 enters the bloodstream after binding to the ACE2 receptor expressed on an endothelial cell of a blood vessel. The virus causes endothelial dysfunction, which results in vasoconstriction and induces ischaemia. Oxygen supply to an organ is restricted, which causes anaerobic respiration. This involves the conversion of pyruvate into lactate by LDH. This causes low production of ATP and reduced pH levels, which results in failure of Na+/K+–ATPase pumps and Ca2+–ATPase pumps. Consequently, sodium ions, calcium ions and water molecules are retained within the tissue, which leads to swelling. In response to ischaemia, reperfusion occurs in which excessive ROS are generated. A cytokine storm is initiated, which involves the JAK–STAT pathway because more IL-6 molecules are activated through STAT3 and vice versa. This results in a positive feedback loop. CRP is then released to the site of inflammation through IL-6 signalling. CRP binds with FcγRI on antibody IgG for opsonisation. Monocytes present in the bloodstream enter the injured tissue as macrophages to start the process of phagocytosis. However, due to oxidative stress, the macrophages are unable to perform their function properly. The high intensity of inflammation results in organ damage and distant organs are affected too.
High level of lactate dehydrogenase and ischaemia–reperfusion injury regulate the multiple organ dysfunction in patients with COVID-19
https://pmj.bmj.com/content/early/2022/03/22/postgradmedj-2022-141573.long
This can explain why we are seeing so many fatal cardiovascular events post Spike Protein exposure.
I am concerned, as noted above, that we are dealing with an inexorably increasing mechanism of damage, which is fatal to those with existing microvascular occlusion. Simultaneously it induces and/or exacerbates existing microvascular occlusion with each exposure. This may also give us a clue as to the reasons for China’s Zero COVID policy, and why reinfections/reexposures may be very dangerous.
Please watch the video, as it contains a great deal of information. I will be expanding on this mechanism. As always, feedback appreciated.
I don’t look at China’s actions the way you do. I don’t think they stem from an authentic interest in reducing reinfections for the sake of protecting people from injury or further injury. I think China’s government does what it does solely to exert and maintain constant, intensifying, highly-responsive behavioral control mechanisms over the population.
I readily admit that I don’t have a way to test that hypothesis against your hypothesis that they do what they do to reduce exposures and infections.
It is interesting how many articles one can find about how dangerous the spike protein is. But not about the injections that force your body to make them. It is clear that there is an extreme bias in the scientific publications. I rather take the Nobel prize winning drug and the other safe alternatives.