Jun 24, 2022Liked by Walter M Chesnut

you really got it!

thank you for your dedication, continued tireless work and service to humanity. there is God's hand after all and there is always hope!

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Jun 24, 2022·edited Jun 24, 2022Liked by Walter M Chesnut

For most of history, our fate was determined by the family we were born into. But now we have access to tools, ideas, proven frameworks, communities and researchers like Walter to help us rapidly accelerate our understanding of amyloid cascade and how we will continue developing our thinking about it.

We all have two choices: what’s easy or what’s going to help us grow our understanding.

Mindset can be an enemy or an ally. Effort and focus always win at the end.

Easy is the enemy. And so are apathy and convenience. 

Thinking and understanding, freedom to THINK & to UNDERSTAND doesn’t come from easy. Our own brain, perceptions, beliefs sabotage us most of the times we choose to go down the hole of EASY. Freedom to THINK and to understand comes from struggle and growth. It comes by doing something that counts, something that’s meaningful.

Walter, I love and admire you. Consider yourself hugged.

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Ray Peat PhD has several articles on Amyloidosis, misfolded proteins and prion disease in general. There exists some simple mitigating strategies which some here might find helpful if you’ve experienced spike protein infection/transfection.

I have no affiliation, but I’ve been studying his research and self-experimenting with Dr. Peat’s ideas for ten years and found these strategies VERY helpful as I age.

The quote below is from this article.


“Everything that is known about the “always fatal” prionic diseases, the diseases of disturbed protein folding, suggests that they can be avoided and even reversed by systematically reversing the processes that amplify inflammation.

People who take aspirin, drink coffee, and use tobacco, have a much lower incidence of Alzheimer’s disease than people who don’t use those things. Caffeine inhibits brain phospholipase, making it neuroprotective in a wide spectrum of conditions. In recent tests, aspirin has been found to prevent the misfolding of the prion protein, and even to reverse the misfolded beta sheet conformation, restoring it to the harmless normal conformation. Nicotine might have a similar effect, preventing deposition of amyloid fibrils and disrupting those already formed (Ono, et al., 2002). Vitamin E, aspirin, progesterone, and nicotine also inhibit phospholipase, which contributes to their antiinflammatory action. Each of the amyloid-forming proteins probably has molecules that interfere with its toxic accumulation.

Thyroid hormone, vitamins A and E, niacinamide (to inhibit systemic lipolysis), magnesium, calcium, progesterone, sugar, saturated fats, and gelatin all contribute in basic ways to prevention of the inflammatory states that eventually lead to the amyloid diseases. The scarcity of degenerative brain disease in high altitude populations is consistent with a protective role for carbon dioxide.“

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This is one of the most significant essays I have read on Covid. You not only identify the pathology very clearly, you also identify the sauce, prognosis and amelioration of the disease. In short you have saved lives. It is shameful that knowledgeable people have chosen to be silent. Evil wins when good people do nothing. I would only add nattokinase from fermented soy is not only very tasty, it also contains an enzyme that dissolves amyloid protein, along with Lumbrokinase. Virtue has its own reward, my friend.

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I will never trust 90% of the people on this planet again.

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So is does this mean there is a likelihood we have a society of AD zombies in a year or two?

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Jun 24, 2022·edited Jun 24, 2022

I'm not a microbiologist, my day to day work is about very different subject matter, and I am trying to make a full picture what is happening, in my head.


I have two questions, please:

So.... What is unclear to me where exactly is the difference between a naturally occurring Alzheimers Disease (small amounts) and how the large amounts of Spike would make a difference?

Secondly, can you please explain shortly why the massive inflammation is needed to trigger this cascade of amyloidosis? Is that because the body would take care of smaller amounts of superantigens, would not be overwhelmed, and it would not lead to a cascade?

Are there (multiple) tipping points? I'm trying to get my head around the mechanism those tipping point hinge on.


Then an observation: I never bought into the explanation (that they're just "winging it") that some people came up with, for the manufacturers to have very different dose amounts (comparing Pfizer-BioNTech to Moderna).

Then, finally I really, really think someone competent should interview you, or cast these observations into some documentary form for easier digestion.

Thank you, on behalf of everyone. Please watch your six. We can't afford to lose you and your (honest) colleages.

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Thank you SO much for this invaluable work. God bless!

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It is conceivable that the mysterious(or covered up?) hepatitis cases in more and more children around the world might have something to do with hepatic amyloidosis(A primary H.A. case study: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4766262/).

Have they ever done liver biopsy in those children with liver failures/transplants to look for amyloid deposits?

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Important paper along with this other one: https://pubs.acs.org/doi/10.1021/jacs.2c03925

Where they prove in vitro the amyloid creation from the S1 spike protein.

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Excellent hypotheses, continued research, clear explication and frequent publication!

You are a Champion!

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Bravo! And this would also apply to the inflammatory cancer cascade. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8267706/

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Wow!  Wondrous Walter's excellence! 

Although contentious, if graphene oxide could possibly be in the jabs, then this study hailing from 2018 is of interest.                       

"Our work shows that graphene can induce the misfolding of prion protein and may cause the  potential risk to biosystems."

 Environ Res. 2019 Apr;171:1-10. doi: 10.1016/j.envres.2018.12.057. Epub 2018 Dec 28.

How graphene affects the misfolding of human prion protein: A combined experimental and molecular dynamics simulation study

Yongchang Zhu 1, Jingjing Guo 1, Ai Zhang 1, Lanlan Li 2, Xuewei Liu 1, Huanxiang Liu 3, Xiaojun Yao 4

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I believe we are being led to a cure either through group intelligence,synchronicity or a higher spiritual intervention.See my comment on previous article re aspirin and it’s role in AD as researched bu Rush medical centre in Chicago.

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Good case study. At least someone is still doing science.

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Sunday, 6/26/22. There are two full page ads, one in the front section of the newspaper and another in the Parade magazine, entitled Do You Have Heart Failure. Suggest you ask your cardiologist about ATTR-CM (transthyretin amyloid cardiomyopathy). ConnectHeartFailure.com. Brought to you by Pfizer.

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