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The Spike Protein, Rapid Aging and ROS: Finding the Etiology for COVID, Long COVID and Post-COVID Conditions
mRNA PROMOTES AS MUCH ROS AS INFECTION: ROS and Cancer, Neurodegeneration, COPD, Diabetes, CVD and Arthritis
Average ROS (Reactive Oxygen Species) levels at the four timepoints. Data are represented as mean (±SD).
As the above figure demonstrates, the Spike Protein is producing excessive levels of ROS post-vaccination. The same paper shows that those who were unvaccinated and without COVID infection had ROS levels of .885. Post vaccination, as you may observe, the ROS levels are sustained at TWICE baseline.
Of course, ROS levels are also elevated during COVID infection, however the study points out a very interesting finding.
Thus, in the second stage of our study, we investigated ROS 45 days after the second dose, as well as after COVID-19 infection, for all individuals and study groups and found that infection promotes less immune response than a second dose of vaccination, although ROS production was at the same level.
A Preliminary Study about the Role of Reactive Oxygen Species and Inflammatory Process after COVID-19 Vaccination and COVID-19 Disease
What do these elevated levels of ROS mean? AGING. Rapid Aging.
What I find remarkable, and evidence for a further understanding of what I have observed and have predicted all along, is that the Spike Protein is a protein that induces RAPID AGING. Now we have a full understanding of one of its mechanisms.
If we look at what excessive ROS induces, we see EXACTLY what we are observing in those post COVID and post mRNA:
Inflammaging is associated with over-abundance of reactive oxygen species in the cell, which can lead to oxidation and damage of cellular components, increased inflammation, and activation of cell death pathways. This review focuses on inflammaging and its contribution to various age-related diseases such as cardiovascular disease, cancer, neurodegenerative diseases, chronic obstructive pulmonary disease, diabetes, and rheumatoid arthritis.
Let us look at neurodegeneration, and those who are reporting ALS-like symptoms.
Schematic showing the molecular mechanisms underlying Alzheimer’s disease (A) and Parkinson’s disease (B). AD, Alzheimer’s disease; Aβ, amyloid beta plaques; BBB, blood brain barrier; CNS, central nervous system; JNK/p38 MAPK, c-Jun N-terminal kinase/p38 mitogen-activated protein kinase; MMP9, matrix metalloprotease 9; NF-κB, nuclear factor kappa-light-chain-enhancer of activated B; PD, Parkinson’s disease; ROS, reactive oxygen species; SIRT1, sirtuin-1.
Inflammaging and Oxidative Stress in Human Diseases: From Molecular Mechanisms to Novel Treatments
Please read the above referenced paper, as it specifically addresses each of the conditions and diseases listed.
I am working on describing the mechanism of how the Spike, once its “factories” have been established in the body, may be traveling incognito via nanotube from cell to cell, inducing pathogenic ROS.
As always, I cannot thank you enough for your support. I will continue my work.
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