THE SPIKE PROTEIN, PLEURAL AMYLOIDOSIS, MESOTHELIOMA AND RECURRENT COVID INFECTION/RECURRENT SPIKE PROTEIN EXPOSURE
THE RELATION OF THE SPIKE PROTEIN TO ASBESTOS
Amyloidosis is characterized by the extracellular deposition of an amyloid substance, and it is classified into systemic and localized types. In the case of SARS-CoV-2, the amyloid substance is the Spike Protein. Uncanny similarities with amyloid-disease associated blood coagulation and fibrinolytic disturbances together with neurologic and cardiac problems led us to investigate the amyloidogenicity of the SARS-CoV-2 Spike protein (S-protein).
A pleural thickness was found on a routine chest X-ray of an asymptomatic 65-year-old man. He had no previous medical history. He had 80 pack-year smoking habit. His occupation was in the oil stove repair, and he had history of exposure to asbestos. However, upon examination and testing, it was determined to be a case of localized pleural amyloidosis mimicking pleural mesothelioma clinically and radiologically.
THE RELATION OF THE SPIKE PROTEIN TO ASBESTOS
I believe the fibrils that are induced by the Spike Protein of SARS-CoV-2 are behaving in precisely the same etiological fashion as asbestos fibers do in nodular pulmonary amyloidosis.
In a case report, amyloid fibrils were observed electron microscopically along with asbestos bodies in the lung parenchyma around a pulmonary nodule. This case shows that a nodule in nodular pulmonary amyloidosis can grow gradually and suggests the possibility of asbestos fibers as one of the etiologic factors in nodular pulmonary amyloidosis.
COVID MESOTHELIOMA MORTALITY RATE AND THE SPIKE PROTEIN REINFECTION/REEXPOSURE CONCERN
What is most concerning here is that COVID is very dangerous for those with Mesothelioma. Nearly one in five patients with malignant pleural mesothelioma (MPM) at a Barcelona hospital contracted COVID-19 during the pandemic. In addition, those patients suffered a 75% MORTALITY RATE
The COVID outcome for these Mesothelioma patients may foreshadow a grim future for humanity. If continued exposure to the Spike Protein induces a fibrotic, “Mesotheliolic” state, what does that say about the prognosis for repeated infections from and exposures to the Spike Protein of SARS-CoV-2?
Those with advanced amyloidic/mesotheliotic states, such as elite athletes with hypertrophic hearts and Mesothelioma patients with advanced amyloidosis/fibrosis, are poised for the final, fatal denouement of hyperaccelerated amyloidosis/fibrosis that severe COVID brings.
MAKE NO MISTAKE. EACH AND EVERY EXPOSURE TO THE SPIKE PROTEIN SEEDS AMYLOIDIC FIBRILS. AGAIN, IF YOU CANNOT UNDERSTAND CHINA’S EXTREME ZERO COVID POLICY, I SUGGEST YOU CONTINUE TO REREAD THIS POST UNTIL YOU DO.
Clearly, we need answers, or our species may be in serious trouble.
QUESTION: GIVEN THAT 20% OF PATIENTS WITH MESOTHELIOMA CONTRACTED COVID, IS SYMPTOMATIC COVID SOLELY BASED ON EXTANT AMYLOIDOSIS/FIBROSIS IN A PATIENT?
Referenced/Related Papers
https://onlinelibrary.wiley.com/doi/10.1111/j.1440-1827.1996.tb03535.x
https://pubmed.ncbi.nlm.nih.gov/10846552/
https://www.biorxiv.org/content/10.1101/2021.12.16.472920v1
https://jtd.amegroups.com/article/view/6501/pdf
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Dear Walter maybe it might be of some interest to you that there is somebody else on substack that looks into Spike & Dementia: https://doorlesscarp953.substack.com/p/pathophysiology-of-spike-protein?s=w
"How do we limit damage caused by disrupting the MAPK pathways, notibly p38? Hydroxychloroquine is one answer and it's a great one: Molecular Insights into the MAPK Cascade during Viral Infection: Potential Crosstalk between HCQ and HCQ Analogues https://www.hindawi.com/journals/bmri/2020/8827752/"
Also, FYI University of Tasmania Australia offers a lot of funding for research into the treatment/causes/care for people in a state of dementia. They are the leading University in Australia focusing on Dementia related research and funding. Maybe you might be interested in contacting them. They also have a world first open course on understanding dementia. https://www.utas.edu.au/wicking/understanding-dementia. Apart from University of Tasmania there are Cognitive cliniques and research centres in Sydney, if you are interested I might be able to connect you with somebody for more information.
On a different note, Ms Kats on twitter did something unacceptable related to your account. On twitter if you take an account and then change the account handle the old comments direct to the new account. In this case the account Dmitry took was twitter.com/parsifaler1 which now doesn't exist but the handle was changed to twitter.com/hom3team which is another alt directing people to his website. Hence the prior @ handles direct to a different handle. An example here https://twitter.com/MsKaywoo/status/1517723958078218246. Approximately, three weeks ago I alerted Twitter and lodge the complaint but still no reply. Most people who follow you are aware of this by now but I am just hurt that it is taking Twitter so long to address such an unacceptable practice.
Given your suspicion do you think China's approach is correct?