If you want to know the future, look at the past. – Albert Einstein
This may explain what embalmers have been reporting re: long white fibrous clots in veins.
Thank you for your work and dedication. Really.
That last paper linked is pretty damning regarding amyloidosis and the damage incurred.
Cntl-F on the word "vaccine" produces zero results, not even a casual mention when discussing potential spike exposure. It's like these guys live in a make-believe world where the vax doesn't exist. I get it as they don't want their careers destroyed, but it's a blatantly glaring omission that harms their credibility.
Totally agree. Signs and symptoms for amyloidosis are prevalent. Patients are showing extreme fatigue. Tests for cardiac tolerance show no explanation. I think different tests are required.
You've hit the nail on the head Walter, excellent work.
Compassionate extinction plan.. Wow
It's like being in a movie
Sophie snd Per wrote about the s-protein in the illness but it was crickets on the same protein in the shots.
I could insert sarcasm here but sometimes it just feels so sad that science is attenuated.
At least it was published.
einstein was a faker, but that saying is quite true. who knows where he stole it :).
Joining the choir and thanking you for your dedication and all your work for humanity. i did my small part and bought a subscription.
Thank you for the history lesson and new information. Look forward to reading the newly published article.
How do we counter all this damage?
How prevalent and persistent in the environment is the spike protein? Is it getting into water supply for instance. It is certainly airbourn from the recently vaccinated.
Any connection to cellulitis? I’ve got a triple vaxxed woman who has cellulitis in her lower leg after a very small puncture wound from a minor strike to her leg by her dog.
Soon you will be back at twitter !
For those of us who have no chemistry knowledge, would some of you who understand this research tell us if the damage is permanent, progressive or just present during illness.
The paper quoted at the bottom shows amyloid was created after exposure to an enzyme secreted by neutrophils in vivo. It is an intriguing hypothesis that this might cause disease however there are some questions that arise:
Why haven't biopsy samples of vaxxed people shown amyloid protein visible under red conga stain in concentrations that match amlyoidosis and in all the usual organs affected by amlyoidosis? It is fairly simple to stain tissue and flouresce it. If amyloid proteins were causing these symptoms we would be able to isolate them in every single tissue sample. Is that the case?
Why do the documented cases of vax induced hepatic liver disease reported go into remission after treatment with steroids. Amyloidosis is cumulative, the proteins reside permanently in your organs and the disease progresses. Steroids may reduce symptoms but don't cause amyloidosis to go into remission.
The paper "Prion-like Domains in Spike Protein of SARS-CoV-2 Differ across Its Variants and Enable Changes in Affinity to ACE2" describes prion sites on the spike which seems similar to what is reported in the amyloidogenesis paper. The conclusion "However, the emerging Omicron (B.1.1.529) variant, although known for its high transmissibility, for now, appears to be milder than previous strains and has the lowest LLR among all SARS-CoV-2 variants." from this paper would seem to me that the risk of amyloid generation is much lower for omicron vs alpha with delta being the highest. Thus any amyloidosis danger is likely behind us as the virus is evolving away from this. Note I believe the amyloidogenesis paper used the alpha amino sequences however I am not sure.
Great work Walter
I don't think that all the RNA to protein translation rules are known for N-1 methylpseudouridine, so Va could be generating slightly different spike proteins with even more potential for harm.
Even for natural RNA nucleotides, software is often needed because of all the special rules.
About 40% of the RNA in the Vs are incomplete fragments.
They could generate even more harmful, shorter proteins.
Would a2 milk be okay for people affected by casein?