SPED AND THE INDUCTION OF GRAFT VS HOST DISEASE (GVHD): SPIKE PROTEIN MOLECULAR MIMICRY OF MHC (HLA): IS LONG COVID GVHD?

The Molecular Mimicry induces HLA Antibodies which initiate GVHD - Why transplants are being rejected.

Please carefully review the above graphic. IT IS LONG COVID!

Graft vs Host Disease occurs as a reaction to a HEMATOPOIETIC STEM CELL TRANSPLANTATION. The Donor’s immune cells attack the hosts tissues. But, you are correct in stating that the Spike Protein is not a stem cell transplantation. Indeed, it isn’t – AND IT IS!

How?

GVHD occurs because the donor’s Major Histocompatability Complex is viewed as foreign to the host. This can induce, among other complications, ANTI-HLA (MHC) ANTIBODIES.

But, how can the Spike Protein induce these antibodies?

BECAUSE THE SPIKE PROTEIN HAS MHC-LIKE DOMAINS. VIA MOLECULAR MIMICRY IT FUNCTIONS AS A HEMATOPOIETIC STEM CELL TRANSPLANTATION!

If the S mutation site was in the MHC-like domain, the mutation enhanced the MHC decoy function. Then the human immune system hard to neutralize these MHC-like sites by producing antibodies. Otherwise, it would affect the normal MHC antigen presentation function by combing MHC and the antibodies. Based on this principle, we analyzed several significant variants that were now popular to determine the MHC-like enhanced region of the S protein.

COVID-19: Attacks Immune Cells and Interferences with Antigen Presentation through MHC-Like Decoy System
https://chemrxiv.org/engage/api-gateway/chemrxiv/assets/orp/resource/item/6108fd830321148575b994b9/original/covid-19-attacks-immune-cells-and-interferences-with-antigen-presentation-through-mhc-like-decoy-system.pdf

GVHD requires existing tissue damage, cytokine release and an activated immune system. In GVHD the conditioning of chemotherapy or radiation causes this tissue damage, cytokine release and activated immune system. With Long COVID, SPED replaces the conditioning of chemotherapy or radiation.

GvHD Advances in Prevention and Treatment of GvHD Webinar Clip

Indeed, we are seeing HLA Antibodies in COVID.

We found that the majority of tested CP volunteers, who recovered form SARS-CoV-2 infection, have developed HLA-Abs. The antibodies were directed against both class I and class II HLA molecules.

SARS-COV-2 Triggers the Development of Class I and Class II HLA Antibodies in Recovered Convalescent Plasma Donors
https://www.karger.com/Article/FullText/524016

This is extremely complicated, which is why it has been so difficult to see. It is a deeply hidden tactic of this monstrous protein. As Edric, the Third Stage Guild Navigator says to the Emperor in Dune: “I see plans within plans.”

Endothelial cell (EC) activation has been suspected of triggering a group of rare and dismal complications that can occur after allogeneic hematopoietic stem cell transplantation (HSCT). Capillary leak syndrome, engraftment syndrome, transplant-associated microangiopathy, diffuse alveolar hemorrhage, and idiopathic pneumonia syndrome are the main nosological entities. Post-HSCT endotheliitis can be triggered by chemotherapy, infections, and calcineurin inhibitors, but allogeneic reactivity is claimed to be the common denominator. Endothelial damages are thought to activate several deleterious pathways (proapoptotic, procoagulant, proinflammatory) and can lead to multiorgan failure; however, clinical manifestations of each syndrome overlap, and their relationship with graft-versus-host disease could be minimal.

Allogeneic reactivity–mediated endothelial cell complications after HSCT: a plea for consensual definitions
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6693000/

This may also explain the myriad of autoimmune disorders being observed post Spike Protein exposure.

I believe this will be a watershed to discovering therapeutics. Of course, if true, will Long COVID be like Chronic GVHD?

I certainly hope not.