Aug 4, 2022·edited Aug 4, 2022Liked by Walter M Chesnut

Thank you Walter. It must be devastating for you to stay focused on such a toxic processes(on so many levels) that enabled all this to happen. I feel sick to my stomach every time I read how deep the pathology runs...and in what ways somebody engineered it. I guess the most terrifying "thing" is that it was enabled by a group of scientist, regulators, medical professionals and government officials. I feel overwhelmed with the anatomy of evil inherent in bioscience, medical institutions, regulators and governments. Your ability to stay with it, with such a focus and perseverance leaves me breathless. Thank you. (The potential for extraordinary and deeply humane is also there...)

On a side note...not sure if you are aware of AbCellera Biologics "Trianni transgenic mouse platform" (see below) Many investment firms ( i.e ARK & Seeking Alpha's ) would be willing to engage& generously remunerate someone like yourself to comment on the below and similar:

"AbCellera Biologics (NASDAQ:ABCL) has entered into a partnership with Atlas Ventures to discover therapeutic antibodies for up to three drug targets.

Terms call for Atlas to have the right to develop and commercialize therapeutic antibodies resulting from the collaboration. AbCellera (ABCL) will receive research payments and is eligible for milestone payments and royalties on sales of products.

AbCellera's (ABCL) technology is based on its Trianni transgenic mouse platform. Atlas is a biotech venture capital firm".

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Aug 4, 2022Liked by Walter M Chesnut

Both ozone and UVB cause lipid peroxidation - the main difference being the amount of antioxidants and ROS produced.

“UVB irradiation induces lipid peroxidation and reduces antioxidant enzyme activities”


“Ozone causes lipid peroxidation but little antioxidant depletion”


What conditions do we have at altitude - high UVB and low oxygen - those are the environmental conditions produced by the spike protein!

Why are athletes more at risk of sudden death? - because their highly metabolic cardiac cells/mitochondria are under so much stress from both the spike protein and the exercise, they release a ton of UV (biophotons) and ROS/free radicals and blow the casket (heart)

And all the medications found to be effective in covid have a great capacity to absorb UV light and therefore reduce this “Stress”

eg Quercetin

“Inhibition of UVA and UVB radiation-induced lipid oxidation by quercetin”


It is this primordial REDOX balance in life/nature, between UVB and Oxygen that controls the actions of an aerobic cell - evolution has simply allowed adaptation on a multicellular scale.

As Above So Below!


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Aug 4, 2022Liked by Walter M Chesnut

It's worse than I thought. Wow.

It's like I've been saying, though. The lethal stage involves ferroptosis and parthanatos. Many other aspects of the virus induce damage by radicals while cutting off endogenous antioxidants.

My map of severe COVID-19 always looked like this: Calcium overload/Nrf2 Suppression/Bradykinin Storm > Nitric Oxide bioavailability loss/eNOS decoupling > Neutrophilia/NETosis > Iron dysbiosis > Fenton Reagent > Hydroxyl Radical formation > Runaway lipid peroxidation/DAMP accumulation/Autoantibodies against OSEs/Ferroptosis/Parthanatos. We can add Spike-induced separation of lipids to that list.

The mid-course is kind of like Lupus, in that Neutrophil Extracellular Traps (or NETs) just keep churning out damaging radicals until antibodies against oxidized lipids and their epitopes (Oxidation-Specific Epitopes, or OSEs) start to form. Oxidized lipids are treated by the immune system as non-self molecules. They're basically foreign objects, like anything else that would trigger inflammation.

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Aug 4, 2022Liked by Walter M Chesnut

Thank you, Walter! What an excellent investigation!

I’m thinking about EBV, that has recently been linked to MS (and also to cancer) and the possibility of EBV reactivation, after infection and/or vaccines, as a causative factor in all this process. Also, EBV has been related to EM/CFS, Fibromyalgia and … amyloidosis.

MCAS is also triggered by it, so what is the role of EBV in all this neuro-circulatory-immune mess?

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Aug 4, 2022·edited Aug 4, 2022

Thank you, Walter. I was just browsing the numerous articles out on pubmed regarding Asherson. Many report occlusions in small and large vessels, but don't really suggest anything other than traditional clotting mechanisms addressed by heparin therapies. If I understood your previous suspicions correctly, you were thinking along the lines that Spike resulted in the formation of amyloid plaques, a completely different process that produces the long, white, rubbery "clots" encountered by morticians and pathologists like Dr. Ryan Cole.

How does Ashersons Syndrome relate to amyloidosis, or do they have separate origins?

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Would lipid removal cause an increase of LDL? I’ve noticed a few people, cholesterol is relatively high in the long COVID/vax injured crowd?

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Walters doing a great job pointing us in the right direction, creating a small community of problem solvers. I take sterolins and beta-carotene and listen to the healers. We should also recognize moral injury to us. Those who survive WW2 recognized moral injury as a way to fortify the soul.

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Thank you. Gosh, after reading this as a layperson it occured to me that, apart

from all other issues, we will need an army of top notch medical scientists

to clear this mess up. And can not be sure of the supportive and honest cooperation of the original perpetrators.

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Aug 4, 2022·edited Aug 4, 2022

Dr Chestnut are you aware that "they" are now debunking Alzhiemers amyloid beta plaque theory? Seems to me that it is possibly being done on purpose, especially if it is potentially a side effect of the gene therapy needle injection. Link Dr Chris Martenson of Peak Prosperity you tube channel https://youtu.be/XnSknwZc9Io

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Sars-Cov-2 = M. avium!

That is why clarithromycin and azithromycin gave such good results in treating Covid-19. M. avium can be accompanied by passenger mycoplasma (Mycoplasma pneumoniae, Chlamydia pneumoniae). M. avium subsp. paratuberculosis and M. bovis causes the prion diseases. Sars-Cov-2 is an astrobiological pathogenic agent belonging to comet Encke (meteor showers, Perseids, Taurids bring this pathogenic agent from the atmosphere to the surface; using the HeLa cell/liquid crystal technology, certain modifications to the genome were transmitted to the mycobacterium in the atmosphere, that is how D614G emerged and also B.1.1.7 and B.1.351).



Dr. Lawrence Broxmeyer did isolate M. avium in patients with Covid-19, back in the spring of 2020:

https://www.academia.edu/43416919/How_BCG_Vaccination_Trials_Might_Finally_Unlock_the_Many_Mysteries_of_COVID_19_ (pgs 9-12)

Sars - bacterial origin:


Sars-cov-2 - bacterial origin:


M. avium and HIV:



M. bovis, Sars-Cov-2, Mers-Cov-2/Omikron, Prions, M. avium/M. paratuberculosis, heat shock proteins (HSP), HeLa cells, Pseudouridine

Sars-Cov-2 and M. bovis:



Heat shock proteins = prion proliferation



Prions = M. bovis = M. paratuberculosis






Mers-Cov and HSP70







Sars-Cov-2 and HSP60, HSP70, HSP90






HSP90 and Pseudouridine


The pseudouridylation of mRNA by PUS 7 increases during heat shock, because the protein moves from the nucleus to the cytoplasm.



CSP (cold shock protein)


HeLa cells and Pseudouridine





M. paratuberculosis and prions





M. avium hsp70




M. paratuberculosis and hsp70





saccharomyces cerevisiae and chlamydia pneumoniae





At the time, the official line in China was that atypical pneumonia, as it was then called, was caused by a Chlamydia bacterium, says Yang Ruifu, a soft-spoken microbiologist and a member of the team at the Academy of Military Medical Sciences (AMMS) that discovered the coronavirus.

In some few sections, coronavirus-like particles were concurrently seen. A coronavirus RNA- polymerase segment (440 bp) was amplified from the lung tissues of two cases of the SARS. After inoculated with materials from the lung samples, the similar Chlamydia-like particles were also found in the inoculated 293 cells. Since the Chlamydia-like agents visualized in both organs and cell cultures could not react with the genus specific antibodies against Chlamydia and monoclonal antibodies against C. pneumoniae and C. psittaci, the results might well be suggestive of a novel Chlamydia-like agent. Since the novel Chlamydia-like agent was found co-existing with a coronavirus-like agent in the dead cases of SARS, it looks most likely that both the agents play some roles in the disease.



Despite these advances, we have a limited understanding of how these modifications exert their functions.

Non-coding RNA and Sars-Cov-2






Non-coding RNA and M. tuberculosis:




Non-coding RNA and prions:





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What kinda of APL treatments are available? Are there any helpful supplements?

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Aug 4, 2022·edited Aug 4, 2022

Fenofibrate protects lipoproteins from lipid peroxidation: synergistic interaction with alpha-tocopherol


Liothyronine (T3 thyroid hormone) seems to help upregulate mitochondrial biogenesis while decreasing Reactive Oxidative Species ROS, both in patients with mtDNA mutations and normal people.

Effect of thyroid hormone on mitochondrial properties and oxidative stress in cells from patients with mtDNA defects


Lipid oxidation that is, and is not, inhibited by vitamin E: Consideration about physiological functions of vitamin E


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About Acuitas lipids;

The lipids used in the Pfizer and Curevac Genetic Vaccine's are produced by Acuitas and is in essence the same as the MC3 lipids they used in Onpattro the first ever RNA drug to be approved.

(The big difference beeing the potency of the payload)

(Still in Investigational category meaning Experimental)

Same goes for the Pfizer Biontech Gene Vaccine.

Here is some very interesting information regarding Onpattro & from onpattro package insert;

From the Norwegian Medicine Agency

Patisiran (Onpattro)

Type of method: Drug; Gene therapy

Area: Neurology

Active substance: Patisiran

Description of the new method;

Patisiran is a siRNA (small interfering RNA) that binds to and inactivates specific messenger RNA, and potentially

blocks the production of transthyretin (TTR), a transport protein for vitamin A and thyroxine, which is produced

mainly in the liver. Patients with hereditary (hereditary) transthyretin amyloidosis (hATTR) have inherited mutations in

the gene that encodes TTR that causes the protein to misfold and form deposits in various tissues and organs (amyloidosis).

By preventing TTR from being produced, patisiran will reduce the accumulation of such deposits in the body (2, 3, 4).

Patisiran is administered as an intravenous infusion and is believed to be indicated for the treatment of patients with hereditary

transthyretin amyloidosis (hATTR) (1, 2)

Nr. 1

Legal presidence for an approved RNA drug as Gene Therapy.

Nr. 2

Recomendations and Adverse Reactions;


You should not use Onpattro if you are pregnant, unless advised by your doctor.

If you are of childbearing age and intend to use Onpattro, you should practise effective contraception.


Ingredients of Onpattro may pass into breast milk. Talk to your doctor about stopping breast-feeding or treatment with Onpattro.

Children and adolescents

Onpattro is not recommended in children and adolescents under 18 years of age.

Likhet mellom dette og C19 injeksjoner;

Side effects;


The most commonly reported adverse reactions have included infusion-related reactions such as flushing, back pain, nausea, abdominal pain, dyspnea, and headache.[Ref]


Atrioventricular heart block was reported (2.7%) of patients; in 3 cases it was a complete AV block.

Common (1% to 10%): Atrioventricular heart block

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Through the history, people used the same strategies to "find out about things". Everything that matters take time, focus and research (asking questions, critical thinking etc) I accept you might have different views and it is totally ok. This is not something that calls for a debate.

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Hi. Thank you! I'm not a scientist but I read many papers and saw you on Gigohmbiologic. The work you are doing is profound. I have a serious set of questions for you and anyone who may know the answer. I've heard leukemia wards have immune compromised people and some habe a sign by their doorr stating they do not want visitors who've had a flu/pneumonia vaccine in the past two weeks. This would be due to it being shed from their bodies. If vaccines do that and the SARS-CoV-2 mRNA treatment is a vaccine then one could assume this may also be thebcase. Now, if you are non vaccinated person and must work and work in a room with people who may be newly vaccinated, is that a recipe for disaster? it seemed like it in my environment in early 2021 and then agagin in fall 2021 when people got their boosters. I think Dr. Kevin McCain is correct and has been since Feb 2020 with his advice about this disease and its treatment. The whole time he has said to mitigate your exposure. Is the risk greater from the vaccinated because we are told they have 12-250x the number of spike protein as an infected unvaccinated individual? Aren't both the strains in the bivalent vaccine a wrong course because the disease has morphed into the 4-5 variant making it like going to get the 2020 flu shot? i expect another major morph in a few weeks after the bivalent release. Last question for you and everyone because I have mental alarm bells flashing and the fog horn blaring. A bivarient SARS-CoV-2 vaccine + a quadravalent influenza vaccine + the live vaccinia vaccine + the US population that may have 50-80% of the people with a compromised immune system = a vastly overwhelmed heath care system that don't even know what they don't even know because they are not doing this immune-calculus. To them its dumb to even ask. My 50-80% number is based on the transfected believing they are correct and the others ar doomed and the non-transfected thinking the same is true of those transfected. Thank you to anyone who answers my questions. I'm just trying to get informed about the actual science which is not the one being propagated mainstream.

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I'm not a medical person. So are the symptoms of "spike protein poisoning" similar to multiple sclerosis, lupus, Bechet's disease, Sjogren's syndrome, systemic sclerosis?

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