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Friday Hope: Vitamin D Revisited: Vitamin D, Calcium Homeostasis and Prevention of Fatal Cardiac Arrhythmias
Could those suffering the recent appearance of excessive Sudden Cardiac Deaths be Vitamin D deficient?
Given my recent work noting the “calcium tsunamis” caused by the Spike Protein, inducing fatal arrhythmias, I looked for reasons for and ways to counteract this occurrence. Interestingly, I found a compound which is helpful for BOTH calcium homeostasis and prevention of fatal cardiac arrhythmias.
One can find abundant evidence for the efficacy of Vitamin D in treating COVID. However, as with many other compounds, there may be additional, critical mechanisms at work making certain treatments even more important than we may initially realize. Vitamin D may be one of the supreme “wonder drugs” for COVID.
It happens to be the case that VITAMIN D DEFICIENCY IS A MECHANISM FOR THE DEVELOPMENT OF FATAL ARRHYTHMIAS.
The levels of serum vitamin D appear to influence cardiovascular risk, and the mechanism involved is linked to the transient outward current and the ultrarapid delayed rectifier K+ current densities, activated through the nuclear vitamin D receptor and Akt pathway. A significant number of studies have correlated vitamin D deficiency with an increased risk of developing cardiac arrhythmias and sudden cardiac death. For this reason, the purpose of this review is to analyze the relation between vitamin D deficiency and the pathogenesis of cardiac arrhythmias. Atrial fibrillation, increased QT interval, and QT dispersion were the most common findings associated with vitamin D deficiency.
Even more interesting, and relevant, is that VITAMIN D allows for the reabsorption of CALCIUM which may mitigate the nanotubular-mediated “calcium tsunamis” of the Spike Protein (please see my previous post).
Vitamin D has many functions, playing an important role in the absorption of calcium and phosphorus, resorption, mineralization and maturation of bone, as well as tubular reabsorption of calcium and its analogues.
Additionally, Vitamin D modulates calcium release, which may also mitigate the observed, fatal “calcium tsunamis.”
Moreover, a heart-failure (HF) rabbit model by Hanafi et al. showed that the vitamin D metabolite 1,25- dihydroxyvitamin D reduces action potential duration and increases left atrium (LA) contractility, possibly via modulation of calcium release.
The Pathogenesis of Cardiac Arrhythmias in Vitamin D Deficiency
Given recent papers an findings, Vitamin D deficiency may be playing a part in those suffering from the recent surge in Sudden Cardiac Deaths. I believe further investigation and studies into these mechanisms and their relation to COVID and the Spike Protein should be conducted ASAP.
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