Many thanks for all the work you are doing. I salute you an many others who are working tirelessly to expose the threat of the virus, the deleterious effects of the vax and the perpetrators of this crime against humanity. God speed!.
Ummm the SARS2 damage in "Long" covid, and the jabs include the mitochondrial function, too. That results in difficulty, often great difficulty, processing oxygen and making sufficient ATP. If the damage is too much, exercise will worsen everything and do more damage (ME/CFS). Exercise will be dependent on the extent of the damage, extent of recovery, managing all the immune issues. It isn't a matter of will, it is physiological. Now that we know more, and have some "spike detox" protocols, I think the injured and impaired will be better situated to get to more regular exercise, the joys it brings and more function.
Another delicious delight from Walter Chesnut! THANK YOU!
How do we reconcile this comment about zinc and the study, for example, cited below?
https://scivisionpub.com/pdfs/covid19-rna-based-vaccines-and-the-risk-of-prion-disease-1503.pdfJ Bart Classen"Zinc ions have been shown to cause the transformation of TDP-43 to its pathologic prion configuratiom. The folding of TDP-43 and FUS into their pathologic prions confirmations is known to cause ALS, front temporal lobar degeneration, Alzherimer'sm and other neurological diseases."---------------------------------------------------------------
Neprilysin and Amyloid Beta Peptide Degradation Author(s): Louis B. Hersh and David W. Rodgers
Neprilysin is a zinc metalloendopeptidase with relatively broad substrate specificity. The enzyme is localized to the plasma membrane of cells where it can function to degrade extracellular peptides. Structural studies show that neprilysin preferentially cleaves peptides on the amino side of hydrophobic amino acids. Neprilysin has been implicated in the catabolism of amyloid β peptides in the brain and as such has received considerable attention, particularly as a therapeutic target for Alzheimers disease. An inverse relationship between neprilysin levels and amyloid β peptide levels and between neprilysin levels and amyloid plaque formation has been observed in human brain. Neprilysin levels decline with aging in the temporal and frontal cortex possibly contributing to higher amyloid β peptide levels. A number of studies have shown that increasing neprilysin levels in the brain leads to a decrease in brain amyloid β peptide levels. Most recently a potential relationship between amyloid β peptide synthesis from the amyloid precursor protein and neprilysin activity has been proposed.
Thank you for your enlightening work. And with the vax they are now suggesting to avoid exercise for a week or more. All those poor people who played regular sports and the problems they are now living with....
CONFIRMATION OF SPARTACUS' FINDING THAT REGULAR EXERCISE MEDIATES DELETERIOUS EFFECTS OF SARS-CoV-2 AND ITS SPIKE PROTEIN
"Regular" is a better word than "chronic" exercise.
Many thanks for all the work you are doing. I salute you an many others who are working tirelessly to expose the threat of the virus, the deleterious effects of the vax and the perpetrators of this crime against humanity. God speed!.
Ummm the SARS2 damage in "Long" covid, and the jabs include the mitochondrial function, too. That results in difficulty, often great difficulty, processing oxygen and making sufficient ATP. If the damage is too much, exercise will worsen everything and do more damage (ME/CFS). Exercise will be dependent on the extent of the damage, extent of recovery, managing all the immune issues. It isn't a matter of will, it is physiological. Now that we know more, and have some "spike detox" protocols, I think the injured and impaired will be better situated to get to more regular exercise, the joys it brings and more function.
https://brandnewtube.com/watch/dr-richard-fleming-how-the-vaxx-is-breaking-down-the-body_O6Vy9D1XVXc4Fn5.html
At 10:27 Dr. Fleming begins to discuss treatment plans. Your thoughts on his recommendations?
Thank you. Muchly.
Another delicious delight from Walter Chesnut! THANK YOU!
How do we reconcile this comment about zinc and the study, for example, cited below?
https://scivisionpub.com/pdfs/covid19-rna-based-vaccines-and-the-risk-of-prion-disease-1503.pdfJ Bart Classen"Zinc ions have been shown to cause the transformation of TDP-43 to its pathologic prion configuratiom. The folding of TDP-43 and FUS into their pathologic prions confirmations is known to cause ALS, front temporal lobar degeneration, Alzherimer'sm and other neurological diseases."---------------------------------------------------------------
Neprilysin and Amyloid Beta Peptide Degradation Author(s): Louis B. Hersh and David W. Rodgers
Neprilysin is a zinc metalloendopeptidase with relatively broad substrate specificity. The enzyme is localized to the plasma membrane of cells where it can function to degrade extracellular peptides. Structural studies show that neprilysin preferentially cleaves peptides on the amino side of hydrophobic amino acids. Neprilysin has been implicated in the catabolism of amyloid β peptides in the brain and as such has received considerable attention, particularly as a therapeutic target for Alzheimers disease. An inverse relationship between neprilysin levels and amyloid β peptide levels and between neprilysin levels and amyloid plaque formation has been observed in human brain. Neprilysin levels decline with aging in the temporal and frontal cortex possibly contributing to higher amyloid β peptide levels. A number of studies have shown that increasing neprilysin levels in the brain leads to a decrease in brain amyloid β peptide levels. Most recently a potential relationship between amyloid β peptide synthesis from the amyloid precursor protein and neprilysin activity has been proposed.
Thank you for your enlightening work. And with the vax they are now suggesting to avoid exercise for a week or more. All those poor people who played regular sports and the problems they are now living with....