Sitemap - 2022 - WMC Research

Friday Hope: Rethinking Prevention

GAME CHANGER: Is Long COVID an Endothelial Disease of the Spike Protein which also Infects Monocytes Inducing Accelerated Glycolysis that Mimics Cancer Cachexia?

A SUGAR-DRIVEN ENDOTHELIAL DISEASE: The Spike Protein, Glycolysis and Methylglyoxal: Endothelial Damage and Cytokine Storms: Why Metformin and Ketogenic Diets Work

Friday Hope and Happiest of Holidays to All: A Fabulous Compendium of Natural Endothelial Protection

The Spike Protein FUNCTIONS AS A “MICROTUMOR”: Instantaneous "Micrometastases": Induction of Tumor Microenvironment: LONG COVID!

Working: Are The Clots We Are Seeing Ocurring Because The Spike Protein Is Turning All It Touches Into Tumor Microenvironment?

Friday Hope: Back to Basics: Vitamin C and the Tumor Microenvironment: Especially in Combination with Fasting-Mimicking Diet

Ivermectin Regulates The Tumor Microenvironment

Is Spike Protein Endothelial Disease (SPED) Actually Part of a More Complex Process? Spike Protein “Metastasis?”

The Paper Published Yesterday by Biering, et al., Demonstrates That the Spike Protein Alone and Independent of ACE-2 Induces Spike Protein Endothelial Disease (SPED)

Friday Hope: Escitalopram as a Possible Therapeutic for COVID/Long COVID and Spike Protein Endothelial Disease (SPED)

The Spike Protein’s Downregulation/Mimicry of GABA May Exacerbate Spike Protein Endothelial Disease (SPED) and Induce Autoantibodies: The Interference With GABA Signaling via Endothelial Interaction

A New Long COVID Study Shows That Depression Is a Major Risk Factor for Long COVID. It Is Also a Major Cause of Endothelial Dysfunction!

All Roads Lead From SPED: Cancer, Fibrosis and Neurodegeneration All Induced By SPED

Friday Hope: Fostamatinib/R406 Blocks Fc Receptor Signaling and Reduces Immune Complex-Mediated Inflammation

Lee, et al, Demonstrate how the Spike Protein AND Its Immune Complexes Are Responsible for the Microvascular Damage (SPED) and Brain Stem Sudden Deaths

Transdifferentiation: The Spike's Cellular Shell Game?

Friday Hope: Another Study Shows Metformin's Benefits Treating COVID - I Propose Its Endothelial/Sympathetic Benefits May Be Its Most Significant

Major Study Being Presented Next Week, Which I Believe Will Strongly Support the SPED Hypothesis

The Cases Of Vaccine Lung Transplant Rejection As Cases Of Advanced Instances Of SPED: Why COVID/Long COVID/SPED Mimics Graft vs Host Disease

Friday Hope: Vitamin D Revisited - A New COVID-19 Study Shows Additional Promise and It Has Anti-Fibrotic Properties: Could Its Suppression Of RAS Be Its Main Benefit?

An Experimental Model for Systemic Organ Fibrosis after SARS-CoV-2 Coronavirus Infection

An Update: Variations on a Theme: Building on CHF and Rabbit Coronavirus

My Current Assessment of SARS-CoV-2: It Is One Of Ralph Baric's Experimental Congestive Heart Failure Coronaviruses That Was Being Further Researched In Wuhan

Friday Hope: Curcumin - The Wonder Polyphenol: A Multifaceted Potential Spike/SPED Therapeutic

A Brainstem Catecholamine Neuron Endgame?

Friday Hope: Melatonin Supresses Catecholamine Synthesis

Ketogenic Diets Deplete Epinephrine. The Aged Have Massive Amounts Of Circulating Catecholamines Compared To The Young. Catecholamine Excess States ARE The Severe COVID Risk States.

Body on Fire: Microvessels are Not Innervated

Bringing it all together. SPED, Catecholamines and Amyloidosis

Is it all SPED and Catecholamines?

Continuing on our recent journey: An important finding related to SPED.

Absolute Bafflement. Great Concern. 150 Dead of CARDIAC ARREST. I believe it could be the Catecholamine Surge. But, what set it off? Working.

Friday Hope: GABA as a Potentially Powerful Therapeutic and its Relation to SPED/Sympathetic Overdrive: Hope for Long COVID

Synthesis of My Previous Two Posts: An Explanation for the Recent Adult "SIDS"

The Catecolamine Surge Induced by the Spike Protein

THE AMYGDALA, SYMPATHETIC OVERDRIVE AND ELEVATED CATECHOLAMINE LEVELS: THE SPIKE PROTEIN’S INDUCTION OF LONG COVID AS A CONSTANT STATE OF “FEAR OR FLIGHT” RESPONSE

TRANSDIFFERENTIATION, DEDIFFERENTIATION, CELLULAR PLASTICITY, DYSPLASIA AND METAPLASIA: IS THE SPIKE PROTEIN CHANGING CELLULAR IDENTITY? COULD CARVACROL AND THYMOL BE POWERFUL THERAPEUTICS?

A CEREBROGENIC HYPOTHESIS FOR THE OBSERVED NON-MYOCARDITIS SUDDEN CARDIAC DEATHS

THE SYSTEMIC REMOVAL OF DEFENSES TO AGE-RELATED DISEASE: THE INDUCTION OF AUTOABS TO AND DOWNREGULATION OF ACE2 (CARDIOVASCULAR DISEASE) AND HLA (AUTOIMMUNITY): INTERACTION WITH p53 (CANCER)

WORKING: WHY CD4 COUNTS ARE MYSTERIOUSLY LOW: AUTOIMMUNITY AND ONCOGENESIS: SPIKE MOLECULAR MIMICRY INDUCING HLA ANTIBODIES: HYPERACCELERATED "HIV"

SPED AND THE INDUCTION OF GRAFT VS HOST DISEASE (GVHD): SPIKE PROTEIN MOLECULAR MIMICRY OF MHC (HLA): IS LONG COVID GVHD?

SPED AS PRELUDE TO A METAPLASTIC DISEASE: PULMONARY, HEPATIC, GASTROINTESTINAL AND CARDIAC COVID FINDINGS

IS SPED JUST THE FIRST PHASE OF A METAPLASTIC DISEASE?

FRIDAY HOPE: A STUDY POSTED OCTOBER 3rd VALIDATES THE SPED HYPOTHESIS - AND OFFERS AN ADDITIONAL THERAPEUTIC - WHICH HAS A NATURAL ALTERNATIVE I PREDICTED WOULD BE BENEFICIAL

A NEW STUDY SHOWING LUNGS AGE ~15 YEARS POST-COVID. SPED EXPLAINS THE FINDING.

IS DEFIBROTIDE AN IMPORTANT TREATMENT FOR SPED, COVID AND LONG COVID?

THE SPIKE PROTEIN’S EFFECT ON THE MICROVASCULATURE OF THE HEART AND OTHER ORGANS IS EQUIVALENT TO A SILENT “HEART ATTACK.” ADDITIONAL DAMAGE FROM ISCHEMIA-REPERFUSION INJURY.

IS THE SPIKE PROTEIN INDUCING “PANCREATIC CANCER” WITHOUT THE TUMOR? IN PANCREATIC CANCER, ONLY 5% OF CELLS ARE TUMOR CELLS, THE REST ARE THE MICROENVIRONMENT

THE PURPOSE OF THE FURIN CLEAVAGE SITE AND THE GP120 INSERT IS TO INDUCE SYSTEMIC FIBROSIS AND CANCER VIA THE ENDOTHELIUM: SPED GP120 AND FURIN. THE FURIN FEEDBACK LOOP. FIBROSIS AND ONCOGENESIS.

WORKING: THE AUTOIMMUNITY (AND PRION DISEASE) BEING OBSERVED POST COVID-19 AND SPIKE PROTEIN EXPOSURE MAY BE DUE TO THE INJURY CAUSED BY THE SPIKE PROTEIN (S1 UNIT IN PARTICULAR)

LIES. IT WAS NEVER “PNEUMONIA.” COVID-19 ARDS IS DUE TO DAMAGED PULMONARY CAPILLARY ENDOTHELIUM: THE IMPORTANCE OF GROUND GLASS OPACITIES (GGO)

SOLUTIONS: ON ENDOTHELIAL PROTECTION

PAPER SUPPORTING SPED HYPOTHESIS POSTED YESTERDAY: CIRCULATING SPIKE FOUND POST COVID AND IN PASC: NO SPIKE FOUND IN UNVACCINATED WHO NEVER HAD COVID

SOLVED: SPED (SPIKE PROTEIN ENDOTHELIAL DISEASE) AND SUDDEN DEATHS: ‘DEAD IN BED’ SYNDROME: THE DAMAGED ENDOTHELIUM CAUSING SYMPATHETIC OVERACTIVITY

IS THE SPIKE PROTEIN BINDING METALS IN OUR CELLS? IS THE SPIKE “CHELATING” THE BODY OF ESSENTIAL BIOMETALS? IS THIS WHY PRION DISEASE IS RAPIDLY PROLIFERATING?

A STARTLING DISCOVERY: IRON OVERLOAD AND “IRRADIATION” (OXIDATIVE) DAMAGE: THE SPIKE PROTEIN MIMICS MECHANISMS OF THE SPACE ENVIRONMENT

EVIDENCE MY SPED "IRRADIATION" HYPOTHESIS IS CORRECT

THE SPIKE PROTEIN’S MIMICKING OF ENDOTHELIAL IRRADIATION AND STEREOTACTIC RADIOSURGERY: THE HIDDEN TIMEBOMB?

LONG COVID (PASC) SLEEP DISORDERS AND SPED – THE MICROVASCULAR ETIOLOGY

SPED AND THE OBSERVED CACHEXIA IN LONG COVID

IS THE SPIKE PROTEIN TRANSFORMING THE VASCULATURE INTO "TUMOR BLOOD VESSELS?"

AND THEN THERE WERE NONE: THE SPIKE PROTEIN’S INEXORABLE DESTRUCTION OF THE MICROVASCULATURE AND THE PATHOPHYSIOLOGY OF COVID, LONG COVID AND SADS

WHY YOUNG, HEALTHY ATHLETES ARE NOT BEING RESUSCITATED. THE MICROVASCULATURE HAS BEEN DESTROYED.

UPDATE: SPIKE PROTEIN ENDOTHELIAL DISEASE (SPED): THE INVOLVEMENT OF THE MITOCHONDRIA: WHY METFORMIN WORKS: WHY SUGAR IS DELETERIOUS

I AM EXPANDING THE SPED HYPOTHESIS TONIGHT. I AM HONESTLY TERRIFIED ABOUT NEW JABS. SUDDEN AND UNEXPECTED DEATH DUE TO BRAIN STEM MICROVASCULAR INJURY.

IT WAS KNOWN IN 2005 THAT THE SPIKE PROTEIN WORSENS ACUTE LUNG FAILURE – AND THAT BLOCKING THE RENIN-ANGIOTENSIN PATHWAY ATTENUATES THE DAMAGE! QUERCETIN, POMEGRANATE PEEL, RUTIN AS THERAPEUTICS

SPED (Spike Protein Endothelial Disease) AS CAUSE OF THE VAST MAJORITY OF THE RECENT SUDDEN CARDIAC DEATHS: DESTRUCTION OF THE BRAINSTEM AND THE CARDIAC MICROVASCULATURE

WHY THE INCREASINGLY LETHAL DAMAGE IS SO DIFFICULT TO FIND AND WHY IT CAN TAKE SO LONG TO MANIFEST: THE SPIKE PROTEIN, ENDOTHELIAL ASSAULT, VASCULITIS, AND THE TRASH COMPACTOR SCENE FROM STAR WARS

THE SPIKE PROTEIN AS HUMAN “SYSTEMIC HERBICIDE”: MITOCHONDRIAL DYSFUNCTION: PHOTOSYNTHESIS AND ATP

FURTHER UNIFICATION OF SPIKE PROTEIN MICROVASCULAR DISEASE AND TAUOPATHY: TLR-2

THE INVISIBLE SPIKE PROTEIN PANDEMIC: THE INTERPLAY OF MICROVASCULAR DESTRUCTION AND ANTIPHOSPHOLIPID SYNDROME

A DARK STORM GATHERING: DISTURBING PARALLELS BETWEEN THE MEASLES N PROTEIN AND THE SARS-CoV-2 S PROTEIN: SUBACUTE SCLEROSING PANENCEPHALITIS

PROOF OF MY LONG COVID AUTOIMMUNE MECHANISM HYPOTHESIS PUBLISHED AUGUST 10th: THE SPIKE INDEED TRAVELS VIA EXTRACELLULAR VESICLES ON ITS OWN

NEW EVIDENCE SUGGESTING THE INVOLVEMENT OF ANTIPHOSPHOLIPID SYNDROME IN LONG COVID

RECURRENT EPISODES OF BINGE EATING IS LIKELY THE MOST IMPORTANT FACTOR IN THE INDUCTION OF OBESITY SYNDROME

OBESITY IS AN INCURABLE DISEASE, PARALLEL TO AUTOIMMUNITY

UNITY: COVID AND PASC IS A B CELL DYSCRASIA: BRINGING ANTIPHOSPHOLIPID SYNDROME AND AMYLOIDOSIS TOGETHER

COVID-19: EXACERBATION AND INDUCTION OF ANTIPHOSPHOLIPID SYNDROME

ALL HYPOTHESES, INCLUDING THOSE POSTED THROUGH TWITTER, NOW ARCHIVED AT WMCRESEARCH.ORG

LIPID REMOVAL, PEROXIDATION AND SYSTEMIC MYELIN DESTRUCTION: THE SPIKE PROTEIN, COVID-19, PASC, DEMYELINATION AND ANTIPHOSPHOLIPID SYNDROME

COVID-19 AND PASC: A DISEASE OF DEMYELINATION - AN UPDATE ON A UNIFYING THEORY OF SPIKE PROTEIN PATHOLOGY

SPIKE PROTEIN INDUCED CYTOKINES AND B-CELL DYSREGULATION: ARE B CELLS OVERPRODUCING MONOCLONAL ANTIBODIES CAUSING AMYLOIDOSIS, AUTOIMMUNITY AND THE REPORTED BLOOD HYPERVISCOSITY?

MORE EVIDENCE PROVING MY EARLY HYPOTHESES CORRECT - HOW MANY LIVES COULD HAVE BEEN SAVED?

BURNING THE CANDLE AT BOTH ENDS: THE SPIKE PROTEIN’S INDUCTION OF AUTOIMMUNITY AND AMYLOIDOSIS AS BOTH PATHOLOGICAL AND AS THE BODY’S PROTECTIVE RESPONSE

ARE COVID AND LONG-COVID COMPLETELY A B-CELL MEDIATED AUTOIMMUNE DISEASE? IS INNATE IMMUNITY THE ONLY TRUE DEFENSE? AND IS INNATE IMMUNITY DIMINISHED WITH EACH EXPOSURE TO THE SPIKE PROTEIN?

DOES THE SPIKE PROTEIN ABROGATE CENTRAL TOLERANCE IN HIGH-AVIDITY AUTOREACTIVE B-CELLS VIA PI3K ACTIVATION

JANUS AND THE FOUR HORSEMEN OF THE SPIKE PROTEIN: COVID, PASC AND DE NOVO “UNEXPLAINED” PATHOLOGIES ALL CAUSED BY THE SPIKE PROTEIN AND ITS IMMUNE COMPLEXES

FURTHER EVIDENCE PROVING MY HYPOTHESIS THAT THE CLEAVED S1 UNIT CAUSES COVID/PASC (SYMPTOMS?) POST mRNA COVID VACCINES

THE FOUR HORSEMEN OF THE SPIKE PROTEIN

A Brief Update On My Spike Protein Progressive Autoimmune Disease Hypothesis

J’ACCUSE! THE SPIKE ANTIBODY/FC-RECEPTOR COMPLEX IS SOURCE OF SEVERE COVID AND THE INDUCTION OF SYSTEMIC AUTOIMMUNE DISEASE: ONE SIDE OF THE AUTOIMMUNE/AMYLOIDOSIS SARS-CoV-2 COIN - AND IT WAS KNOWN!

END STAGE COVID IS ASHERSON'S SYNDROME - EARLY COVID MIMICS CHURG-STRAUSS: MY WORK FROM DECEMBER 2020

URGENT WARNING! THE SPIKE PROTEIN AS SOURCE OF CONSTANT INJURY AND CHRONIC INFLAMMATION: ALL ROADS LEAD TO AIDS AND THE “FIBROSED” THYMUS: THE IMMUNE SYSTEM AS THE BUS IN "SPEED"

OF BATS, CORONAVIRUSES AND EXTREME HUMAN (INFLAMM)AGING: WHY BAT CORONAVIRUSES ARE HUMAN NEUTRON BOMBS: MCC950 TO THE RESCUE

LONG COVID IS A PHENOTYPE OF THE OLDEST OLD INDUCED BY THE SPIKE PROTEIN: AT THE NEXUS OF CACHEXIA AND AMYLOIDOSIS

UNDERSTANDING THE CNS AND PERIPHERAL NERVOUS SYSTEM PATHOLOGY OF COVID AND LONG COVID FROM AN ALZHEIMER’S PERSPECTIVE: A HYPERACCELERATED HYBRID OF ALZHEIMER’S (AD) AND PARKINSON’S DISEASE (PD)

THE SPIKE PROTEIN, THE AMYLOID CASCADE AND INFLAMMATION: FOLDING SPACE TO RAPID SYSTEMIC AMYLOIDOSIS VIA INFECTION OR VACCINE

PILOTS, PASC AND THE SPIKE PROTEIN AMYLOIDOSIS HYPOTHESIS

SADS, MALE INFERTILITY AND THE SPIKE PROTEIN AMYLOIDOSIS HYPOTHESIS

Now Appearing on Kevin McCairn's YouTube Stream

COVID-19 IS NOT A RESPIRATORY OR VASCULAR DISEASE. IT IS A DISEASE OF RAPID AND INDOLENT AMYLOIDOSIS.

QUERCETIN AND CURCUMIN AS POTENTIALLY POWERFUL "PRIOLOID" THERAPEUTICS

BNT162b2, ANCHORLESS PRIONS AND CEREBRAL AMYLOID ANGIOPATHY

THE “PRIOLOID” SPIKE PROTEIN OF SARS-COV-2 AND FATAL TRANSMISSIBLE AMYLOID ENCEPHALOPATHY

SEVERE COVID, LONG COVID AND SPIKE-PROTEIN INDUCED SYSTEMIC AMYLOIDOSIS AS AN AUTOCATALYTIC REACTION: IT IS NOT A QUESTION OF AMYLOID OR PRION, IT IS DEPENDENT ON THE RATE OF PROTEIN AUTOCATALYSIS AND

SEVERE COVID AND LONG COVID AS RAPID AND INDOLENT VARIANTS OF SYSTEMIC FIBRINOGEN AMYLOIDOSIS

WHY RAY LIOTTA, AND MILLIONS OF OTHERS, ARE DYING IN THEIR SLEEP - SYSTEMIC AMYLOIDOSIS

AMYLOID FIBRIL FORMATION AS MEANS OF VIRAL INFECTION ENHANCEMENT: A CONNECTION WITH HIV

AN INFILTRATIVE DISEASE, AN ALZHEIMER’S OF THE BODY: IS INFILTRATION OF THE SARS-CoV-2 SPIKE PROTEIN INTO ORGAN TISSUE THE CAUSE OF LONG COVID? AND DOES IT AWAIT US ALL?

TRANSMISSIBILE AMYLOIDOSIS: “WE CAN INDUCE THE FULL-BLOWN DISEASE JUST BY ADMINISTERING THESE PROTEIN AGGREGATES”

Absolute Confirmation of my Spike Protein Amyloidosis Hypothesis

CONFIRMATION OF SPARTACUS' FINDING THAT REGULAR EXERCISE MEDIATES DELETERIOUS EFFECTS OF SARS-CoV-2 AND ITS SPIKE PROTEIN

THE SPIKE PROTEIN AND EXPERIMENTAL PRODUCTION OF AMYLOIDOSIS IN HUMANS

SPIKE PROTEIN INDUCED AMYLOIDOSIS AS ETIOLOGY FOR OBSERVED CARDIAC PATHOLOGIES

COVID-19, REDRUM AND MURDER

AMYLOIDOSIS AS ETIOLOGY OF SEVERE COVID AND LONG COVID

SERUM AMYLOID A AND FELINE INFECTIOUS PERITONITIS

ARE THE CASES OF "HEPATITIS" IN CHILDREN ACTUALLY SPIKE PROTEIN AMYLOIDOSIS OF THE LIVER?

"CYANIDE" SELF-POISONING

A HIGHLY TRANSMISSIBLE ZYKLON-B

THE SPIKE PROTEIN, PLEURAL AMYLOIDOSIS, MESOTHELIOMA AND RECURRENT COVID INFECTION/RECURRENT SPIKE PROTEIN EXPOSURE

MINOCYCLINE, RECENTLY ADOPTED BY CHINA TO TREAT COVID-19, IS A POTENT ANTI-AMYLOID

ASP: SPIKE PROTEIN AMYLOIDOSIS

FURTHER CONFIRMATION OF MY HYPOTHESIS THAT THE SPIKE PROTEIN INDUCES A “DNA DISEASE”

THE ULTIMATE DANGER OF THE SPIKE PROTEIN

THE SPIKE PROTEIN IS THE “AMYLOID” BEING DEPOSITED AND INDUCING AMYLOIDOSES: A MAJOR FINDING MISSED

A NEW KIND OF CANCER: THE MARRIAGE OF ONCOGENESIS AND PRIONOPATHY

AN ALZHEIMER’S OF THE HEART

THE SPIKE PROTEIN FORMS COMPLEXES WITH ALL AMYLOIDOGENIC PROTEINS

A UNIFYING THEORY OF COVID-19 PATHOGENESIS, COMBINING SENESCENT, CARDIOVASCULAR, ONCOGENIC AND NEURODEGENERATIVE PATHOLOGIES

SARS-CoV-2 SPIKE PROTEIN AS VIROPORIN, INDUCING BRAINSTEM SPREADING DEPOLARIZATION (SD) RESULTING IN SUDDEN CARDIAC DEATH

SPIKE PROTEIN ACTIVATION OF LINE-1 RETROTRANSPOSONS

“VAIDS” AS REALITY

AFTER DESTROYING THE ENDOTHELIUM, THE SPIKE PROTEIN, IN ADDITION TO INDUCING FIBROSIS, THEN TURNS THE VASCULATURE TO “IRON AND STONE”

SUDEP AND “DIED UNEXPECTEDLY”

CODED TO KILL: A SHORT-RANGE, MID-RANGE AND LONG-RANGE CELLULAR WEAPON

THE SPIKE PROTEIN IS A “TURNKEY SOLUTION” TO INDUCE COAGULATION AND FIBROSIS

DECODING GABRIN’S SIGN IN THE CONTEXT OF COVID

A DEFINITIVE CASE FOR A CONTROVERSIAL THERAPEUTIC

COVID-19: AN IMMUNE MEDIATED DISEASE OF PROGRESSIVE SYSTEMIC FIBROSIS INDUCED BY THE SPIKE PROTEIN AND ITS IMMUNE COMPLEXES

SPIKE PROTEIN FIBROSIS SYNDROME

THE SPIKE PROTEIN SUPERANTIGEN, MYOCARDITIS AND MORTALITY

THE SPIKE PROTEIN CAUSES TO THE EXACT SAME DAMAGE TO THE ENDOTHELIUM AS IF IT HAD BEEN IRRADIATED

HOW THE SPIKE PROTEIN INDUCES MYOCARDITIS

NOT “PNEUMONIA!” GROUND GLASS OPACITIES IN COVID-19

THE MULTISCALE PHATE PAPER AND THE IMMUNE PHENOTYPE OF ADVANCED AGE

A PARRALEL BETWEEN JUVENILE RHEUMATOID ARTHRITIS (JRA) AND THE SPIKE PROTEIN

VITAMIN D, OR VITAMIN D BINDING PROTEIN

THE SPIKE PROTEIN AND HEMATOPOIETIC STEM CELL LYSIS

SARS-CoV-2 AND ITS RELATION TO FELINE INFECTIOUS PERITONITIS

Coming soon