SPED (Spike Protein Endothelial Disease) AS CAUSE OF THE VAST MAJORITY OF THE RECENT SUDDEN CARDIAC DEATHS: DESTRUCTION OF THE BRAINSTEM AND THE CARDIAC MICROVASCULATURE
All COVID-19 patients had microvascular abnormalities on postmortem magnetic resonance imaging (MRI).
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Death due to microvascular occlusion in sickle-cell trait following physical exertion
What I believe we are observing is a parallel to the microvascular occlusion seen in those with sickle cell disease. Except, it is not sickled erythrocytes which are occluding the cardiac microvasculature. It is the Spike Protein damaged endothelium of the microvasculature which has healed, but in this process of healing, has become occluded by FIBROTIC SCAR TISSUE.
This has been observed in autopsies of COVID-19 victims.
The pathogenesis of COVID-19 myocardial injury does not seem to relate to primary myocardiocyte involvement but to local inflammation with associated interstitial edema. We found heightened TGF-β and interstitial collagen expression in COVID-affected hearts, a potential harbinger of chronic myocardial fibrosis. These results suggest a need for continued clinical surveillance of patients for myocardial dysfunction and arrythmias after recovery from the acute phase of COVID-19.
The Pathogenesis of COVID-19 Myocardial Injury: An Immunohistochemical Study of Postmortem Biopsies
As is noted above, it is not PRIMARY MYOCARDIOCYTE INVOLVEMENT. It is the inflammation and injury of the Spike Protein which ultimately causes the fibrosis, leading to potentially fatal arrhythmias.
Please refer to my previous post for the mechanism of this Spike Protein induced feedback loop which ultimately causes microvascular DELETION.
I believe there is another mechanism by which the Spike Protein can cause sudden cardiac death. The IMMUNE COMPLEXES OF THE SPIKE PROTEIN ARE BEING DEPOSITED INTO THE BRAINSTEM CAUSING STRUCTURAL DAMAGE AND ITS DYSFUNCTION.
Platelet aggregates were observed in all COVID-19 cases but were rare in control individuals (mean, 16 vs 0.3 vessels with aggregates/mm2; P <.0001). In COVID-19 cases, more aggregates were observed in the hindbrain (mean, 20 vs 14 vessels with aggregates/mm2; P =.04).
They were also found to have immune complex deposits in all regions of the brain, predominantly in the extracellular matrix and some glial cells and neurons.
And what precipitates all this damage? THE SPIKE PROTEIN’S ATTACK ON THE ENDOTHELIUM. AGAIN, THE ONE-TWO PUNCH - DIRECT ASSAULT AND SUBSEQUENT IMMUNE MEDIATED DAMAGE consistent with my theory of SPED.
“Cytotoxicity directed against the endothelial cells is the most likely initiating event that leads to vascular leakage, platelet aggregation, neuroinflammation and neuronal injury,” the researchers concluded.
Neurovascular Injury Evident in Sudden Death of Patients With COVID-19
This also explains why these young individuals cannot be revived by CPR or defibrillation. The NEURONAL PATHWAYS AND/OR THE MICROVASCULAR VESSELS HAVE BEEN DESTROYED.
This isn’t going away. This isn’t something we can brush aside any longer, nor should have we ever. People are dying. Young people. Needlessly. If the medical community allows this to continue, from this point forward, I view them as murderers.
EXPOSURE TO THE SPIKE PROTEIN OF SARS-CoV-2, ESPECIALLY THE ORIGINAL WUHAN SPIKE, MUST BE TERMINATED IMMEDIATELY.
We need therapeutics and treatments. I have gleaned several possiblities if you scan my Substack. That being said, the most urgent advice would be: IF YOU HAVE STUCK YOUR HAND IN THE FIRE AND IT IS NOW BURNING, TAKE YOUR HAND OUT OF THE DAMN FIRE!