PROOF OF MY LONG COVID AUTOIMMUNE MECHANISM HYPOTHESIS PUBLISHED AUGUST 10th: THE SPIKE INDEED TRAVELS VIA EXTRACELLULAR VESICLES ON ITS OWN
The Spike Protein travels WITHOUT the presence of other viral RNA
A brief post to confirm that my long hypothesized mechanism of autoimmunity in Long COVID (and in the pathology of Long COVID itself) is true.
The Spike Protein indeed travels via extracellular vesicles without the presence of other viral RNA.
Additionally, we report that part of the circulating Spike protein is linked to extracellular vesicles without any presence of viral RNA in these vesicles.
Persistent Circulation of Soluble/EV-Linked Spike Protein and Viral RNA in Individuals with Post-Acute Sequelae of COVID-19
My hypothesis from September 15, 2021:
Once the mitochondria are damaged, this then activates the autoimmune response of the body. The accumulation of defective mitochondria led to overproduction of an inflammatory protein called type 1 interferon. The findings suggest that failed quality control of mitochondria may cause Sjogren’s, lupus, and other autoimmune diseases through production of interferon.
The Spike Protein then proceeds to travel from cell to cell via EXTRACELLULAR VESICLES. This means, of course, they will NOT BE FOUND IN THE BLOOD. We would not have been aware all this time. As the S1 unit has been found in monocytes 15 months post infection, it may be traveling intracellularly, executing a “Sherman’s March Through Georgia” on the mitochondria, resulting in multisystemic autoimmunity.
THE. AUTOIMMUNE. MECHANISM.