33 Comments

A friends wife is suffering from low iron. She's passed out and had 1blood infusion in rhe past month. She isnt making red blood cells like she used to.

2x vaxxed, they can't find root cause. This sounds like the reason.

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“Can’t find the reason” or not acknowledging the fact that these injections are poison. I’m beyond frustrated with these nincompoops! Use your brians! Figure something out ding dongs!

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Alright, all of your comments plus Walter's article here inspired me to look around; I put up a new post relevant to iron transport via Transferrin Receptor, which is a target of spike. The hemoglobin synthesis post isn't ready yet.

https://medquotes.substack.com/p/not-new-spike-transferrin-receptor

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As a J&J vax injured person, this is pretty interesting. I’ve had two blood tests and both show abnormally low iron

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My step-father has had to get several iron injections since receiving the Pfizer shots.

My brother-in-law is injured from the J & J. He’s no longer able to work, but he won’t share any of his health details with anyone but his doctor.

I am beyond frustrated.

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I’m sorry to hear that. Hopefully his doctor is of some help. I only found a flccc doctor somewhat helpful and I’m one of the milder cases (for now).

We’ll only be taken serious in a decade when they figure out ace-2 auto antibodies can be caused by the vax and COVID. 🙄

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If we live that long that is...

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So where is the unused iron going, if it's not being made into blood cells after spike protein exposure? Is it relevant that the solid substances removed from the veins of people who died unexpectedly after their Covid shots, and sent to Mike Adam's lab for mass spec analysis, are full of metals including tin and iron, but not full of blood proteins at all?

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I was most concerned over the biodistribution to the bone marrow due to the fact it could potentially affect all new progenitor cells (hemopoietic) being made by bone marrow. Other places of biodistribution are bad too, regarding potential fertility and organ damage, but if one survives in spite of those issues having the bone marrow affected concerned me that it could result in a never ending assault on cellular renewal affecting the blood, vascular & circulatory systems and many of the biochemical cell signaling cascades!

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I've been following a lady in my Facebook friends. She developed severe anaemia . She has had numerous blood transfusions and iron infusions and Drs still not have found the cause. I asked her if she had had any problems in the past and she hadn't. She was told a few months ago, her levels were so low she was high risk of death. I found a link to a number of people with similar experiences post vax, very low iron levels, never before with problems, now having transfusions and iron infusions, and none with any explanation as to the root cause. She started feeling unwell last September, had a chest infection she couldn't seem to get rid of, then was suffering fatigue, then in November had first blood transfusion. But her last episodes, just never seems to manage to get her levels up :( Like you say a never ending assault.

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I wonder, is it possible the blood transfusions are giving her new spike proteins?

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Oh gawd. Crikey imagine if? Could it? I mean I totally believe there is something in the vaccine shedding phenomenon after what I experienced last Summer being around a newly vaccinated man. Hell I've had slipped discs and had back problems on and off for a few years now, but going numb in my backside was a new one for me. Lasted about 2 months then vanished and not had any problems since, and the vaxed man vanished too .LOL. I remember plenty of vexed people refusing to eat in same restaurants as untaxed, well I'm telling you I don't particularly want to get intimate with a freshly vaxed man anymore.

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SARS-CoV-2 Infects Red Blood Cell Progenitors and Dysregulates Hemoglobin and Iron Metabolism

https://pubmed.ncbi.nlm.nih.gov/35181867/

Acquired aplastic anemia following SARS-CoV-2 vaccination https://pubmed.ncbi.nlm.nih.gov/35592930/

Severe aplastic anemia after COVID-19 mRNA vaccination: Causality or coincidence?

https://pubmed.ncbi.nlm.nih.gov/34920343/

These authors are in Arizona in case anyone needs to be seen there:

Severe Aplastic Anemia After Receiving SARS-CoV-2 Moderna mRNA Vaccination

https://pubmed.ncbi.nlm.nih.gov/35356634/

These authors are in Texas:

SARS-CoV-2 infection associated with aplastic anemia and pure red cell aplasia

https://pubmed.ncbi.nlm.nih.gov/35452511/

These authors are in Louisville:

An evidence that SARS-Cov-2/COVID-19 spike protein (SP) damages hematopoietic stem/progenitor cells in the mechanism of pyroptosis in Nlrp3 inflammasome-dependent manner

https://pubmed.ncbi.nlm.nih.gov/34163002/

Acquired Aplastic Anemia as a Clonal Disorder of Hematopoietic Stem Cells

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8219510/

These authors are in Wuhan:

A New Immunosuppressive Therapy for Very Severe Aplastic Anemia in Children with Autoantibodies

2022. https://pubmed.ncbi.nlm.nih.gov/35258748/

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There was a Swedish study early November 2021. Game changer in molecular biology. The vaccine or virus spike protein enters the nucleus of the cells and down regulates DNA repair. Merogenomics C-19 vaccine update 18 YouTube.

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More TLR4 news. There are methods to add to vaxx recovery protocols to modify and interfere with adverse action there. Hope you can get the attention of medical doctors reading this who can explore further.

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NATURE "In addition to these two receptors, SARS-CoV-2/COVID-19 may also interact with the extracellular matrix metalloproteinase inducer basigin, known as cluster of differentiation 147 (CD147) as well as recently proposed with C-type lectin receptor and Tweety family member 2"

https://web.archive.org/web/20220117202805/https://www.nature.com/articles/s41375-021-01332-z

Is there a way to explain what "Tweety family member" means in more of a dinner with the neighbors type description than uber Chestnut science?

It's more curiosity about terminology. In the 1990's bone marrow transplants evolved to stem cell transplants, then T-cell transplants which were called "A Buffy Coat" by HUTCH team.

It hasn't crossed my mind since until the "Tweety family" showed up and made me wonder if it's connected. Buffy & Tweety sounds like a good pair to me.

Thanks for doing what you do & God bless all the good hearted brainiacs! <3

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Thank you Walter, I have shared this.

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I know of someone in his sixties that had to take iron post vaccination because he felt fatigued, questioned if it was usual for a male?

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The body sequesters iron when fighting inflammation.

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It seems to me that humanity has become the lab rat for mRNA injectables. This is the sense I am getting from all responses above. I also believe that mRNA that was first injected was no longer the same in the booster. Why? I am not any kind of a health practitioner. Just scratching my head and posting out loud. Because the symptoms and reactions change with each booster. Also as the age groups become younger we see different reactions to the jab. This is an experiment by pharma paid for us with our taxes, health and ultimately our lives.

What is the true objective? What are pharma learning by tweaking the mRNA injectables? Still scratching my head???

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There are reports that boosters contained stronger dosage, perhaps double.

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I have a couple friends that say their first two shots were fine, they felt absolutely nothing. In both cases it was the Booster which made them feel AWFUL.

Humans being used like Lab rats.😔

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The body's immune response changes each time, and the immune system of younger people functions differently than older people. Response likewise depends upon previous encounters with similar antigens. This uncertainty has been discussed for many years, which is one reason why these didn't come to the market sooner, but conveniently took stage during an emergency.

In a practical sense, this is why one cannot rationalize having had a mild response to the first dose, and therefore expect the same outcome next time.

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Thank you. What are your insights into mRNA. It is made as I understand it from a computer model of the Sars Cov2 spike protein. That means that it is relatively easy to tweak the model for different batches of injectable mRNA lots. I suggest this, because the FDA allowed I believe Pfizer to forgo extensive test as the current mRNA injectables are accepted and the same development process will be followed. So essentially no testing.

With the following in mind;

1) no long term studies of future mRNA injectables (at least Pfizer),

2) The ease at which the mRNA can be changed without notice (see 1 above)

3) Pfizer and other Pharma mRNA injectables refuse to allow independent assay of the product and they refuse to provide a list of ingredients. Informed consent is no longer an option... We the taxpayer did pay for this and we have a right to know what the ingredients are or if they are changing them and why we don't have the raw data feeds of adverse events or on the safety tests pharma did run. Why?

4) Quality control is a continuing issue.

and

And as you point out it is dose dependent. Each jab will be a new adventure. Why then all the secrecy regarding lot numbers, ingredients, reporting of adverse events etc. How is it possible to create a injectable mRNA jab when the target Sars Cov2 is rapidly mutating. Seems that something beside profit is being explored at the taxpayers expense. Since the CDC and Pharma are not letting the public see the raw data that is coming in it gives me pause. I don't have a warm fuzzy feeling about any of this.

What are your speculations or insights into the on going saga of EUAs

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It sounds like you have a good sense of the situation.

Here's a post discussing some of these questions:

https://boriquagato.substack.com/p/are-covid-boosters-causing-more-severe

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Actually don't know much as we are not told much. Considering almost two years now, I don't know much and what little I know is learned here, from David Martin, Robert F. Kennedy jr., Dr. Dr. McCullough, Dr. Malone, and many others, as well as continuing data analysis by Jessica Rose, Swiss Policy Research, Jennifer Margulis and more.

All this info has been gotten by pulling teeth so to speak. All I am getting is the crumbs from all the hard work of others and their possible prosecution and harassment by Government and the media.

I appreciate the link and the work you and all who share info, remaining diligent and healthy.

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Here is a link from Swiss Policy Research. They are very conservative in their posting, very informative.

https://swprs.org/more-injections-more-infections/

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They are Trying to figure out how they, the elites, can escape most illnesses so THEY can LIVE A LONG TIME and not worry about heart disease, cancer etc. getting them till they are 200 years old!

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Maybe.

It may also be a means to facilitate Transhumanism. To augment humans with devices needs a means to prevent rejection of said device by the subjects immune system. It could be they are injecting these mRNA payloads to develop ways to manipulate the immune system to allow augmentation. In the process many humans my die. Those left will be the ones that can be modified.

Next in line please1???

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Yes, and M-CSF is activated by UVB and Vit D

“UVB‐induced GM‐CSF production is suppressed by dexamethasone in HaCaT Cells”

https://www.researchgate.net/publication/227684171_UVB-induced_GM-CSF_production_is_suppressed_by_dexamethasone_in_HaCaT_Cells

https://www.researchgate.net/publication/262228981_Erratum_Vitamin_D_endocrine_system_and_osteoclasts

high M-CSF serum levels significantly correlated with high 25-hydroxyvitamin D3 serum levels suggesting that 25-hydroxyvitamin D3 might modulate M-CSF release

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4144809/

And so is HIF-1

“UVB radiation induces expression of HIF-1alpha and VEGF through the EGFR/PI3K/DEC1 pathway”

https://pubmed.ncbi.nlm.nih.gov/16964427/

“Calcitriol Suppresses HIF-1 and HIF-2 Transcriptional Activity by Reducing HIF-1/2α Protein Levels via a VDR-Independent Mechanism”

https://pubmed.ncbi.nlm.nih.gov/33182300/

Light controls life!

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Autoimmunity is related to Th1/Th2 and Th17/Treg balance - which is related to the balance between UVB and Ozone!

UVB inhibits Thl responses and stimulates Th2 responses

https://repub.eur.nl/pub/20439/001025_Boonstra,%20Pieter%20Andre.pdf

Whereas, ozone can have the opposite effect:

“Th1 cells activation might be predominant over Th2 activation upon ozone exposure in asthmatic children”

https://pubmed.ncbi.nlm.nih.gov/24641287/

https://www.nature.com/articles/pr2009119/

UVB also reduces Th17

https://pubmed.ncbi.nlm.nih.gov/24617827/

And increases Tregs

leading to a restoration of the balance between Th17 and Tregs with a shift towards the Tregs side.

https://onlinelibrary.wiley.com/doi/10.1111/exd.12369

UVB also activates PI3K and CREB

https://pubmed.ncbi.nlm.nih.gov/11965545/

Which in turn activates c-Fos

https://www.sciencedirect.com/science/article/abs/pii/S0378111902007230

c-Fos is increased in SARS-CoV2

https://pubmed.ncbi.nlm.nih.gov/33284859/

C-Fos is involved in cell proliferation, differentiation and survival and its dysregulation is an important factor for cancer development as well as inflammation and autoimmunity.

https://pubmed.ncbi.nlm.nih.gov/32042069/

The PI3K pathway also regulates GSK-3Beta

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3694284/

GSK-3B is a serine-threonine protein kinase involved in several signaling pathways also regulating cell proliferation, differentiation, and apoptosis and is implicated in diabetes, Alzheimer's Disease, Parkinson's Disease, schizophrenia, and some types of cancer.

It is also one of five genes that seem to facilitate infection with SARS-CoV2!

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9014212/

UVB inhibits GSK-3B which plays a role in the increase of COX2

https://pubmed.ncbi.nlm.nih.gov/11389054/

Whereas chronic Ozone exposure increases GSK-3B

https://www.sciencedirect.com/science/article/abs/pii/S0161589018300336

“Chronic O3 exposure induced increases of collagen deposition, MMP8, MMP9, HIF-1α, VEGF, α-SMA, and GSK-3β”

Th17 Polarization also depends on GSK3β and GSK3β Inhibition Enhances Treg Function.

https://www.frontiersin.org/articles/10.3389/fonc.2020.01221/full

GSK-3B also controls PD-1 PDL1 levels

https://pubmed.ncbi.nlm.nih.gov/31604533/

PD-1 regulates the balance between T cell activation, tolerance, and immunopathology!

https://pubmed.ncbi.nlm.nih.gov/18173375/

UVB increases PD-L1

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6548650/

And PD-1/PD-L1 plays a role in the maintenance of UVB-induced tolerance.

https://pubmed.ncbi.nlm.nih.gov/27556047/

GSK-3B is also important for the activation of MITF

“activation of GSK3beta by cAMP facilitates MITF binding to the tyrosinase promoter”

https://pubmed.ncbi.nlm.nih.gov/12093801/

Moreover, the PI3K pathway drives the maturation of mast cells via MITF

https://pubmed.ncbi.nlm.nih.gov/21791431/

And MITF can regulate chemokine expression, immune cell attraction and infiltration.

https://europepmc.org/article/PMC/6290759

“MITF also modulates the expression of coinhibitory receptors, i.e., PD-L1 and HVEM, and the production of an inflammatory secretome, which directly affects the infiltration and/or activation of the immune cells”

https://molecular-cancer.biomedcentral.com/articles/10.1186/s12943-020-01290-7

MITF is activated by UVB

https://onlinelibrary.wiley.com/doi/epdf/10.1111/j.1755-148X.2010.00775.x

MITF is partially located in the mitochondria and regulates PDH.

Overexpression of MITF reduces PDH activity, indicating that MITF is a negative regulator of PDH

https://www.jacionline.org/article/S0091-6749(16)31344-6/fulltext

And, PDH is reduced in SARS-CoV2

https://www.nature.com/articles/s41467-021-22166-4

With a corresponding increase in MITF!

“Bost et al.’s study of SARS-CoV-2 host-viral infection maps identified MITF as one of the up-regulated genes”

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8145325/

So, why is MITF increased in Covid?

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Bost et al: "The gene for MITF lies downstream of M-CSF (macrophage colony-stimulating factor), a cytokine known as a growth factor for differentiation and growth of monocytes and macrophages [36]."

M-CSF is produced by many cell types in response to viral infection

MITF -- HIF1a:

PMID: 15983061: Hypoxia-inducible factor 1α is a new target of microphthalmia-associated transcription factor (MITF) in melanoma cells

Happy hypoxics: Upon infection by human coronavirus SARS-CoV-2, HIF1a is required for induction of glycolysis in monocytes and the consequent pro-inflammatory state (PubMed:32697943). In monocytes, induces expression of ACE2 and cytokines such as IL1B, TNF, IL6, and interferons (PubMed:32697943). Promotes human coronavirus SARS-CoV-2 replication and monocyte inflammatory response (PubMed:32697943).

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“We also demonstrated that another unusual pathway, the calcium-independent PI3K/PKCE/JNK/CREB pathway, functioned in cooperation with the calcium-dependent pathway to induce COX-2 expression upon stimulation by spike protein.”

Just thinking out loud here. Would Cox-2 inhibitors be useful or harmful?

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