Continuing on our recent journey: An important finding related to SPED.
Is a chronic catecholamine storm resulting in heart failure or fatal arrhythmia the "endgame" of repeated Spike exposure?
Working.
Please read the following paper. It outlines the current direction of my research.
Endothelial cells are able to synthesize and release catecholamines both in vitro and in vivo
https://pubmed.ncbi.nlm.nih.gov/22665130/
That makes perfect sense.
Thanks for being a true Scientist with a conscience, compassion and respect for truth instead of an immoral traitor to humanity like so many of the others in your field.
I did a review of the vax initiated Kynurenine pathway and yes, there is a connection:
The Relationship between Kynurenine, Catecholamines, and Arterial Hypertension in Mesangioproliferative Glomerulonephritis
Abstract
In our previous work we proved that hyperkynureninemia can participate in the development of mesangioproliferative glomerulonephritis (MPG) by impairing renal endotheliocytes and epitheliocytes (Rudzite et al., 1991). We have also found close association between serum kynurenine (K) and intensive hematuria and arterial hypertension (AH) in the patients with MPG (Martinsons et al., 1992). Although K is not a vasoconstrictor, it stimulates the effect of serotonin, norepinephrine and acetylcholine in blood vessels (Lapin and Umanskaja, 1980). K activates serum dopamine-beta-hydroxylase in vitro: the concentration of dopamine decreases and that of noradrenaline increases (Rudzite et al., 1987). Such a mechanism in the development of AH has been demonstrated in patients with essential AH (Rudzite et al., 1987).
Keywords
Diastolic Blood Pressure
Arterial Hypertension
Essential Hypertension
Arterial Blood Pressure
Active Amine
https://link.springer.com/chapter/10.1007/978-1-4613-0381-7_64